Optimisation of GWAS p-value threshold.
- J. Kenneth Baillie (177195)
- Andrew Bretherick (4903141)
- Christopher S. Haley (4903156)
- Sara Clohisey (4747563)
- Alan Gray (3276414)
- Lucile P. A. Neyton (4903150)
- Jeffrey Barrett (4903153)
- Eli A. Stahl (229145)
- Albert Tenesa (107674)
- Robin Andersson (3501872)
- J. Ben Brown (4903144)
- Geoffrey J. Faulkner (481019)
- Marina Lizio (185917)
- Ulf Schaefer (237265)
- Carsten Daub (3815110)
- Masayoshi Itoh (15728)
- Naoto Kondo (32868)
- Timo Lassmann (14297)
- Jun Kawai (13865)
- Damian Mole (4903138)
- Vladimir B. Bajic (8687)
- Peter Heutink (39587)
- Michael Rehli (18004)
- Hideya Kawaji (13861)
- Albin Sandelin (2089)
- Harukazu Suzuki (15733)
- Jack Satsangi (259264)
- Christine A. Wells (200908)
- Nir Hacohen (220591)
- Thomas C. Freeman (4903147)
- Yoshihide Hayashizaki (3101)
- Piero Carninci (3015)
- Alistair R. R. Forrest (185918)
- David A. Hume (87441)
Publication date
March 2018
DOIAbstract
<p>Coexpression signals are shown for six different –log<sub>10</sub>(p) bins for GWAS p values from a single study of Crohn’s disease. From each bin, 800 SNPs were selected at random. No signal for coexpression is detected at weak p-values.</p
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