prd1, α-ada, and imac genetically interact during dendrite pruning in ddaC sensory neurons.

(A) Dendrites of imac170/+, prd1M56/Df(3R)Exel7310, and imac170/+; prd1M56/Df(3R)Exel7310 ddaC neurons at 16 h APF. (B) Dendrites of imac172/+, prd1M56/Df(3R)Exel7310, and imac172/+; prd1M56/ Df(3R)Exel7310 ddaC neurons at 16 h APF. (C) Dendrites of α-ada3/+, prd1M56/Df(3R)Exel7310, and α-ada3/+; prd1M56/Df(3R)Exel7310 ddaC neurons at 16 h APF. (D) Dendrites of prd1M56/+, α-ada RNAi #2/+, and prd1M56/α-ada RNAi #2 ddaC neurons at 16 h APF. (E) Dendrites of α-ada3/+, imac RNAi/+, and α-ada3/+; imac RNAi/+ ddaC neurons at 16 h APF. Quantification of total length of unpruned dendrites at 16 h APF. The number of samples (n) in each group is shown on the bars. Error bars represent SEM. Scale bar (A) represents 50 μm. **p p S1 Data. The genotypes can be found in S1 Text. α-ada, α-adaptin; APF, after puparium formation; Df, deficiency; imac, immaculate connections; prd1, pruning defect 1; RNAi, RNA interference.