Figure S2. Disease-specific DEGs clusters samples by phenotype in publicly available expression data. Expression values for the disease-specific genes identified in our analysis were extracted from publicly available datasets: A) GSE1145 (microarray), B) GSE1869 (microarray), C) GSE55296 (RNA-seq), and D) GSE46224 (RNA-seq). (TIFF 15964Â kb
IntroductionHeart failure (HF) is a complex clinical syndrome leading to high morbidity. In this stu...
Figure S3. Comparative cellular component analysis of control and TGF-β1-induced fibrosis models. G...
Data from: "The Reference of the Transcriptional Landscape of Human End-Stage Heart Failure" Source...
Table S3. DEGs for unadjusted gene expression. DEGs at FDR ≤ 0.05 in DCM vs NF and ICM vs NF. (XLSX ...
Table S7. Predicted biofunctions. IPA biofunctions predicted to be increased or decreased for Analys...
Figure S1. Empirical distribution of FDR values â¤0.05 for 1000 permutations. Histogram with the nu...
Table S1. Sample table. Characteristics including sex, race, ethnicity, age, cause of death (if appl...
Table S5. Enriched IPA Canonical Pathways. IPA canonical pathways for p ≤ 0.05 for Analysis 1 (HF-DE...
Table S2. RNAseq quality control table. Raw read counts ranged from ~â36 to 66 million. Read count...
Table S6. Enriched IPA Toxicity. IPA toxicity lists for p ≤ 0.05 for Analysis 1 (HF-DEGs). (XLSX 15 ...
Abstract Background Current heart failure (HF) treatment is based on targeting symptoms and left ven...
ObjectivesThis study was designed to identify a common gene expression signature in dilated cardiomy...
OBJECTIVES: Clinically, patients with chronic heart failure arising from different etiologies receiv...
Heart failure (HF) causes a great socioeconomic and medical burden in our societies. We aimed to scr...
Overlapping genes among significant pathways for hearf failure before data processing. (PDF 145Â kb
IntroductionHeart failure (HF) is a complex clinical syndrome leading to high morbidity. In this stu...
Figure S3. Comparative cellular component analysis of control and TGF-β1-induced fibrosis models. G...
Data from: "The Reference of the Transcriptional Landscape of Human End-Stage Heart Failure" Source...
Table S3. DEGs for unadjusted gene expression. DEGs at FDR ≤ 0.05 in DCM vs NF and ICM vs NF. (XLSX ...
Table S7. Predicted biofunctions. IPA biofunctions predicted to be increased or decreased for Analys...
Figure S1. Empirical distribution of FDR values â¤0.05 for 1000 permutations. Histogram with the nu...
Table S1. Sample table. Characteristics including sex, race, ethnicity, age, cause of death (if appl...
Table S5. Enriched IPA Canonical Pathways. IPA canonical pathways for p ≤ 0.05 for Analysis 1 (HF-DE...
Table S2. RNAseq quality control table. Raw read counts ranged from ~â36 to 66 million. Read count...
Table S6. Enriched IPA Toxicity. IPA toxicity lists for p ≤ 0.05 for Analysis 1 (HF-DEGs). (XLSX 15 ...
Abstract Background Current heart failure (HF) treatment is based on targeting symptoms and left ven...
ObjectivesThis study was designed to identify a common gene expression signature in dilated cardiomy...
OBJECTIVES: Clinically, patients with chronic heart failure arising from different etiologies receiv...
Heart failure (HF) causes a great socioeconomic and medical burden in our societies. We aimed to scr...
Overlapping genes among significant pathways for hearf failure before data processing. (PDF 145Â kb
IntroductionHeart failure (HF) is a complex clinical syndrome leading to high morbidity. In this stu...
Figure S3. Comparative cellular component analysis of control and TGF-β1-induced fibrosis models. G...
Data from: "The Reference of the Transcriptional Landscape of Human End-Stage Heart Failure" Source...