Fibrinogen decreases cardiomyocyte contractility through an ICAM-1-dependent mechanism-0

lipopolysaccharide (LPS) or saline into rats. Acquired with group mean data, the curves demonstrate an LPS-induced increased end diastolic volume (EDV), maintenance of stroke volume (SV), and therefore a marked reduction of left ventricular ejection fraction (SV/EDV). E, end-systolic elastance.<p><b>Copyright information:</b></p><p>Taken from "Fibrinogen decreases cardiomyocyte contractility through an ICAM-1-dependent mechanism"</p><p>http://ccforum.com/content/12/1/R2</p><p>Critical Care 2008;12(1):R2-R2.</p><p>Published online 3 Jan 2008</p><p>PMCID:PMC2374637.</p><p>