Nitric oxide-dependent bone marrow progenitor mobilization by carbon monoxide enhances endothelial repair after vascular injury
- Wegiel, Barbara
- Gallo, David J.
- Raman, Kathleen G.
- Karlsson, Jenny M.
- Ozanich, Brett
- Chin, Beek Y.
- Tzeng, Edith
- Ahmad, Shakil
- Ahmed, Asif
- Baty, Catherine J.
- Otterbein, Leo E.
Publication date
February 2010
DOI Publisher
Ovid Technologies (Wolters Kluwer Health) Language
EnglishAbstract
Carbon monoxide (CO) has emerged as a vascular homeostatic molecule that prevents balloon angioplasty-induced stenosis via antiproliferative effects on vascular smooth muscle cells. The effects of CO on reendothelialization have not been evaluated
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