BV induced ERK phosphorylation only in neurons that did not co-express p-p38 in the rat L4/5 spinal dorsal horn

(A-C) Double immunofluorescence in spinal dorsal horn sections (16 μm) for p-ERK (green) and NeuN, a neuronal marker (red). Double immunofluorescence indicated significant colocalization between p-ERK and NeuN. (D-F) Double immunofluorescence in the dorsal horn of an L4/L5 spinal cord section (16 μm) for p-ERK (green) and p-p38 (red). Double immunofluorescence indicated p-ERK and p-p38 did not colocalize. Scale bars, 50 μm (A-F).<p><b>Copyright information:</b></p><p>Taken from "Differential activation of p38 and extracellular signal-regulated kinase in spinal cord in a model of bee venom-induced inflammation and hyperalgesia"</p><p>http://www.molecularpain.com/content/4/1/17</p><p>Molecular Pain 2008;4():17-17.</p><p>Published online 30 Apr 2008</p><p>PMCID:PMC2391153.</p><p>