Effect of <i>Helicobacter pylori</i> infection on infiltration of immune cell subsets into adipose tissue.
- Josep Bassaganya-Riera (120051)
- Maria Gloria Dominguez-Bello (120056)
- Barbara Kronsteiner (120061)
- Adria Carbo (120066)
- Pinyi Lu (120070)
- Monica Viladomiu (120075)
- Mireia Pedragosa (120080)
- Xiaoying Zhang (52883)
- Bruno W. Sobral (85497)
- Shrinivasrao P. Mane (85493)
- Saroj K. Mohapatra (120090)
- William T. Horne (120096)
- Amir J. Guri (120100)
- Michael Groeschl (120105)
- Gabriela Lopez-Velasco (120109)
- Raquel Hontecillas (120113)
Publication date
November 2012
DOIAbstract
<p>Macrophages (F4/80<sup>+</sup>CD11b<sup>+</sup>) and regulatory T cells (CD4<sup>+</sup>CD25<sup>+</sup>Foxp3<sup>+</sup>) were immunophenotyped in white adipose tissue (WAT) from leptin receptor-deficient (db/db) mice infected with either the wild-type <i>H. pylori</i> 98–325 (white bars), the isogenic <i>H. pylori</i> 99–305 (black bars), or uninfected (dashed bars), (n = 10 mice/group). Statistically significant differences (<i>P</i><0.05) between treatments (*) are indicated.</p
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