<p>Jurkat cells (1 × 10<sup>6</sup>/well) were exposed to DHA (0, 10, 20, 40, 80 μM) or DHA/HTF for 48 h, and measured apoptosis by Annexin V/PI staining and flow cytometry. DHA induced a dose-dependent increase in apoptosis (a). This difference was statistically significant (p < 0.001). DHA/HTF induced a 2.75-fold increase in apoptosis relative to control (p < 0.001). The results represented mean ± standard deviation (b). ***p<0.001, compared with the control group, <b>#</b> p<0.001, compared with DHA.</p
<div><p>(A) DHC increases p53 lysine 373 and 382 acetylation in a dose-dependent manner in the TK6 c...
AbstractDocosahexaenoic acid (DHA) is an omega-3 fatty acid under intense investigation for its abil...
<p>PD98059, an inhibitor of ERK phosphorylation, enhanced DHA-induced decrease of cell viability. Ce...
<p>Jurkat cells (1 × 10<sup>4</sup>/well) were exposed to DHA (0, 2.5, 5, 10, 20, 40, 80 μM) for 24,...
<p>Jurkat cells (1 × 10<sup>6</sup>/well) were exposed to DHA (0, 10, 20, 40, 80 μM) or DHA/HTF for ...
(A) Cell death was evaluated at 24, 48, 72, and 96 h cells dosed with 7 mM DHA. Cells categorized in...
<p>(A) DHA induced activation of caspase-8 and -9 assessed by fluorometric assay. Cells were treated...
<p>Jurkat cells (1 × 10<sup>6</sup>/well) were exposed to DHA (0, 10, 20, 40, 80 μM) or DHA/HTF for ...
<p>(A) DHA-induced concentration-dependent reduction of cell viability in A549 and PC-9 cells. Cells...
<p>Jurkat cells (1 × 10<sup>6</sup>/well) were exposed to DHA (0, 10, 20, 40, 80 μM) or DHA/HTF for ...
<p>Jurkat cells (1 × 10<sup>6</sup>/well) were exposed to DHA (0, 10, 20, 40, 80 μM) or DHA/HTF for ...
<p>Combination treatment with HDACi and DHA increased cell death. Cells were treated with 4 mM NaB, ...
<p>(A) FaDu, Cal-27, and Hep-2 cells were treated with indicated concentrations of DHA for 24 or 48h...
(A) HepG3 cells were exposed to DHA doses ranging from 0–10 mM for 5 days. Cells were counted for ea...
(A and C) Apoptotic markers, caspase 1 and caspase 2 were probed to define the cell death mechanism....
<div><p>(A) DHC increases p53 lysine 373 and 382 acetylation in a dose-dependent manner in the TK6 c...
AbstractDocosahexaenoic acid (DHA) is an omega-3 fatty acid under intense investigation for its abil...
<p>PD98059, an inhibitor of ERK phosphorylation, enhanced DHA-induced decrease of cell viability. Ce...
<p>Jurkat cells (1 × 10<sup>4</sup>/well) were exposed to DHA (0, 2.5, 5, 10, 20, 40, 80 μM) for 24,...
<p>Jurkat cells (1 × 10<sup>6</sup>/well) were exposed to DHA (0, 10, 20, 40, 80 μM) or DHA/HTF for ...
(A) Cell death was evaluated at 24, 48, 72, and 96 h cells dosed with 7 mM DHA. Cells categorized in...
<p>(A) DHA induced activation of caspase-8 and -9 assessed by fluorometric assay. Cells were treated...
<p>Jurkat cells (1 × 10<sup>6</sup>/well) were exposed to DHA (0, 10, 20, 40, 80 μM) or DHA/HTF for ...
<p>(A) DHA-induced concentration-dependent reduction of cell viability in A549 and PC-9 cells. Cells...
<p>Jurkat cells (1 × 10<sup>6</sup>/well) were exposed to DHA (0, 10, 20, 40, 80 μM) or DHA/HTF for ...
<p>Jurkat cells (1 × 10<sup>6</sup>/well) were exposed to DHA (0, 10, 20, 40, 80 μM) or DHA/HTF for ...
<p>Combination treatment with HDACi and DHA increased cell death. Cells were treated with 4 mM NaB, ...
<p>(A) FaDu, Cal-27, and Hep-2 cells were treated with indicated concentrations of DHA for 24 or 48h...
(A) HepG3 cells were exposed to DHA doses ranging from 0–10 mM for 5 days. Cells were counted for ea...
(A and C) Apoptotic markers, caspase 1 and caspase 2 were probed to define the cell death mechanism....
<div><p>(A) DHC increases p53 lysine 373 and 382 acetylation in a dose-dependent manner in the TK6 c...
AbstractDocosahexaenoic acid (DHA) is an omega-3 fatty acid under intense investigation for its abil...
<p>PD98059, an inhibitor of ERK phosphorylation, enhanced DHA-induced decrease of cell viability. Ce...