Deletion of CnB1 in keratinocytes enhances the skin acute inflammatory response to TPA treatment and to UVB exposure.

  • Xunwei Wu (5172356)
  • Alice Tommasi di Vignano (5172362)
  • Qian Zhou (125859)
  • Piotr J. Michel-Dziunycz (5172359)
  • Fuxiang Bai (5172350)
  • Jun Mi (374600)
  • Jing Qin (41397)
  • Tingjian Zu (5172353)
  • Günther F. L. Hofbauer (117408)
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Publication date
May 2018

Abstract

<p><b>A</b>. Ear thickness was measured at different time points as indicated. ## p<0.01 when comparing the TPA-treated group with the untreated group, ** p<0.01 when comparing the CnB1-/- (KO) group (KO+TPA) with the control CnB1+/+ group (Ctrl+TPA); <b>B.</b> Histological analysis of ears of Ctrl and KO mice at 48 hr after TPA treatment; <b>C.</b> Thickness of dorsal skin collected at the indicated time points was measured using a microscope. ## p<0.01 when comparing the UVB-exposed group with the untreated group, ** p<0.01 when comparing the KO group (KO+UVB) with the control group (Ctrl+UVB); <b>D.</b> Histological analysis of dorsal skins of Ctrl and KO mice at 48 hr after exposure to UVB; <b>E-H.</b> Sections from <b>C</b> and <b>D</b...

Extracted data

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