Diesel exhaust exposure favors TH2 cell recruitment in nonatopic subjects by differentially regulating chemokine production.
- Chang, Ying
- Sénéchal, Stéphanie
- De Nadai, Patricia
- Chenivesse, Cécile
- Gilet, Jules
- Vorng, Han
- Legendre, Benjamin
- Tonnel, André-Bernard
- Wallaert, Benoît
- Lassalle, Philippe
- Tsicopoulos, Anne
Publication date
August 2006
DOI Publisher
Elsevier BV ISSN
0091-6749 Citation count (estimate)
22Abstract
The prevalence of allergic diseases has increased in the last 20 years, and a number of studies have shown that diesel exhaust particle-associated polyaromatic hydrocarbons can exacerbate the allergic reaction. Much less is known about their potential capacity to generate a T(H)2-type allergic reaction in nonatopic subjects.Journal ArticleResearch Support, Non-U.S. Gov'tinfo:eu-repo/semantics/publishe
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