The last few years have seen remarkable developments in our understanding of the physiology, pharmacology and genetics of inhibitory glycinergic synapses. In part, this has been due to the development of new resources such as specific antisera recognizing glycine receptor (GlyR) and transporter (GlyT) subtypes, but also the characterization of new mouse, zebrafish and bovine genetic models of glycinergic dysfunction. What is also evident is the high quality and impact of the research conducted in this field. This is reflected in the reviews and research articles in this Special Issue entitled “Glycinergic transmission: physiological, developmental and pathological implications”
Hereditary hyperekplexia is caused by disinhibition of motoneurons resulting from mutations in the i...
Defects in mammalian glycinergic neurotransmission result in a complex motor disorder characterized ...
Glycine transporters (GlyTs) are Na(+)/Cl--dependent neurotransmitter transporters, responsible for ...
Glycinergic neurotransmission is a major inhibitory influence in the CNS and defects are associated ...
Hereditary hyperekplexia is caused by disinhibition of motoneurons resulting from mutations in the i...
Glycine serves a dual role in neurotransmission. It is the primary inhibitory neurotransmitter in th...
Reduced inhibitory glycinergic neurotransmission is implicated in a number of neurological condition...
AbstractTransmitter-gated ion channels mediate rapid synaptic transmission in the CNS and constitute...
Tese de doutoramento, Ciências Biomédicas (Neurociências), Universidade de Lisboa, Faculdade de Medi...
Glycine receptors (GlyRs) are found in most areas of the brain, and their dysfunction can cause seve...
Glycine receptors (GlyRs) are found in most areas of the brain, and their dysfunction can cause seve...
AbstractThe glycine transporter subtype 1 (GlyT1) is widely expressed in astroglial cells throughout...
Glycine is an important inhibitory neurotransmitter that is localized in the caudal areas of the ner...
The glycine receptor chloride channel (GlyR), a member of the pentameric Cys-loop ion channel recept...
The identity of a glycinergic synapse is maintained presynaptically by the activity of a surface gly...
Hereditary hyperekplexia is caused by disinhibition of motoneurons resulting from mutations in the i...
Defects in mammalian glycinergic neurotransmission result in a complex motor disorder characterized ...
Glycine transporters (GlyTs) are Na(+)/Cl--dependent neurotransmitter transporters, responsible for ...
Glycinergic neurotransmission is a major inhibitory influence in the CNS and defects are associated ...
Hereditary hyperekplexia is caused by disinhibition of motoneurons resulting from mutations in the i...
Glycine serves a dual role in neurotransmission. It is the primary inhibitory neurotransmitter in th...
Reduced inhibitory glycinergic neurotransmission is implicated in a number of neurological condition...
AbstractTransmitter-gated ion channels mediate rapid synaptic transmission in the CNS and constitute...
Tese de doutoramento, Ciências Biomédicas (Neurociências), Universidade de Lisboa, Faculdade de Medi...
Glycine receptors (GlyRs) are found in most areas of the brain, and their dysfunction can cause seve...
Glycine receptors (GlyRs) are found in most areas of the brain, and their dysfunction can cause seve...
AbstractThe glycine transporter subtype 1 (GlyT1) is widely expressed in astroglial cells throughout...
Glycine is an important inhibitory neurotransmitter that is localized in the caudal areas of the ner...
The glycine receptor chloride channel (GlyR), a member of the pentameric Cys-loop ion channel recept...
The identity of a glycinergic synapse is maintained presynaptically by the activity of a surface gly...
Hereditary hyperekplexia is caused by disinhibition of motoneurons resulting from mutations in the i...
Defects in mammalian glycinergic neurotransmission result in a complex motor disorder characterized ...
Glycine transporters (GlyTs) are Na(+)/Cl--dependent neurotransmitter transporters, responsible for ...