AbstractThe tumor necrosis factor receptor 1 (TNFR1), a prototypic member of the death receptor family signals both cell survival and apoptosis. In this issue of Cell, Micheau and Tschopp (2003) report that apoptotic TNFR1 signaling proceeds via the sequential formation of two distinct complexes. Since the first complex can activate survival signals and influence the activity of the second complex, this mechanism provides a checkpoint to control the execution of apoptosis
AbstractTRAF proteins are major mediators for the cell activation, cell survival, and antiapoptotic ...
AbstractTNF's main function is to stimulate inflammation by turning on gene transcription through th...
Signals from tumor necrosis factor α (TNFα) are transduced through two types of receptors, tumor nec...
AbstractThe tumor necrosis factor receptor 1 (TNFR1), a prototypic member of the death receptor fami...
AbstractApoptosis induced by TNF-receptor I (TNFR1) is thought to proceed via recruitment of the ada...
AbstractSignaling by receptors in the TNF receptor (TNFR) superfamily mediate biological outcomes ra...
Present adress of Olivier Micheau: INSERM U517, Faculties of Medicine and Pharmacy, 7 Bd Jeanne d'Ar...
AbstractTumor necrosis factor receptor (TNFR) superfamily members can induce a context-dependent apo...
Early events in the signalling of tumor necrosis factor-receptor 1 (TNF-R1), which is the main TNF r...
AbstractLigand binding to tumor necrosis factor receptor-I (TNFRI) can promote cell survival or acti...
Apoptosis induced by TNF-receptor I (TNFR1) is thought to proceed via recruitment of the adaptor FAD...
AbstractThrough its type 1 receptor (TNFR1), the cytokine TNF elicits an unusually wide range of bio...
AbstractDeath domain containing members of the tumor necrosis factor receptor (TNFR) superfamily can...
AbstractFas (APO-1/CD95) and tumor necrosis factor receptor 1 (TNFR1) trigger apoptosis by recruitin...
AbstractMany diverse activities of tumor necrosis factor (TNF) are signaled through TNF receptor 1(T...
AbstractTRAF proteins are major mediators for the cell activation, cell survival, and antiapoptotic ...
AbstractTNF's main function is to stimulate inflammation by turning on gene transcription through th...
Signals from tumor necrosis factor α (TNFα) are transduced through two types of receptors, tumor nec...
AbstractThe tumor necrosis factor receptor 1 (TNFR1), a prototypic member of the death receptor fami...
AbstractApoptosis induced by TNF-receptor I (TNFR1) is thought to proceed via recruitment of the ada...
AbstractSignaling by receptors in the TNF receptor (TNFR) superfamily mediate biological outcomes ra...
Present adress of Olivier Micheau: INSERM U517, Faculties of Medicine and Pharmacy, 7 Bd Jeanne d'Ar...
AbstractTumor necrosis factor receptor (TNFR) superfamily members can induce a context-dependent apo...
Early events in the signalling of tumor necrosis factor-receptor 1 (TNF-R1), which is the main TNF r...
AbstractLigand binding to tumor necrosis factor receptor-I (TNFRI) can promote cell survival or acti...
Apoptosis induced by TNF-receptor I (TNFR1) is thought to proceed via recruitment of the adaptor FAD...
AbstractThrough its type 1 receptor (TNFR1), the cytokine TNF elicits an unusually wide range of bio...
AbstractDeath domain containing members of the tumor necrosis factor receptor (TNFR) superfamily can...
AbstractFas (APO-1/CD95) and tumor necrosis factor receptor 1 (TNFR1) trigger apoptosis by recruitin...
AbstractMany diverse activities of tumor necrosis factor (TNF) are signaled through TNF receptor 1(T...
AbstractTRAF proteins are major mediators for the cell activation, cell survival, and antiapoptotic ...
AbstractTNF's main function is to stimulate inflammation by turning on gene transcription through th...
Signals from tumor necrosis factor α (TNFα) are transduced through two types of receptors, tumor nec...
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