Mice have become a favored species to model disease. Many mouse strains have proven relatively resistant to some manipulations that have generated renal disease in other species. Kirchhoff et al. describe a means of producing hypertension, proteinuria, and glomerular sclerosis in a mouse strain
<div><p>Nephrin (<i>Nphs1</i>) is an adhesion protein that is expressed at the podocyte intercellula...
Podocytes are terminally differentiated and structurally unique cells having interdigitating foot pr...
Nephrin, a major component of the glomerular slit diaphragm (SD), is both a structural protein as we...
Regulation and maintenance of the actin cytoskeleton of podocytes has emerged as a critical process ...
Nephrin in experimental glomerular disease.BackgroundThe recently identified gene NPHS1 with its mut...
Nephrin is a slit diaphragm protein critical for structural and functional integrity of visceral glo...
Nephrin localizes to the slit pore of the glomerular epithelial cellBackgroundRecognition that mutat...
Podocytes are highly differentiated epithelial cells that contribute to the selective filtration bar...
Increasing evidence implicates activation of NF-κB in a variety of glomerular diseases, but the mech...
Mutations in the NPHS2 gene, encoding podocin, are responsible for familial autosomal recessive and ...
The molecular mechanisms maintaining glomerular filtration barrier are under intensive study. This s...
Background: Several studies have shown that activation of the renin-angiotensin system may lead to h...
Nephrin (Nphs1) is an adhesion protein that is expressed at the podocyte intercellular junction in t...
Mutations in the NPHS1 gene cause congenital nephrotic syndrome of the Finnish type. The gene produc...
Downregulation of nephrin in podocytes leads to development of proteinuria in human and experimental...
<div><p>Nephrin (<i>Nphs1</i>) is an adhesion protein that is expressed at the podocyte intercellula...
Podocytes are terminally differentiated and structurally unique cells having interdigitating foot pr...
Nephrin, a major component of the glomerular slit diaphragm (SD), is both a structural protein as we...
Regulation and maintenance of the actin cytoskeleton of podocytes has emerged as a critical process ...
Nephrin in experimental glomerular disease.BackgroundThe recently identified gene NPHS1 with its mut...
Nephrin is a slit diaphragm protein critical for structural and functional integrity of visceral glo...
Nephrin localizes to the slit pore of the glomerular epithelial cellBackgroundRecognition that mutat...
Podocytes are highly differentiated epithelial cells that contribute to the selective filtration bar...
Increasing evidence implicates activation of NF-κB in a variety of glomerular diseases, but the mech...
Mutations in the NPHS2 gene, encoding podocin, are responsible for familial autosomal recessive and ...
The molecular mechanisms maintaining glomerular filtration barrier are under intensive study. This s...
Background: Several studies have shown that activation of the renin-angiotensin system may lead to h...
Nephrin (Nphs1) is an adhesion protein that is expressed at the podocyte intercellular junction in t...
Mutations in the NPHS1 gene cause congenital nephrotic syndrome of the Finnish type. The gene produc...
Downregulation of nephrin in podocytes leads to development of proteinuria in human and experimental...
<div><p>Nephrin (<i>Nphs1</i>) is an adhesion protein that is expressed at the podocyte intercellula...
Podocytes are terminally differentiated and structurally unique cells having interdigitating foot pr...
Nephrin, a major component of the glomerular slit diaphragm (SD), is both a structural protein as we...
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