The human malaria parasite Plasmodium falciparum increases red blood cell membrane permeability during infection to allow for import of nutrients and other solutes. Nguitragool et al. (2011) have now identified parasite-encoded CLAG3 proteins as key components of the import channel located on the erythrocyte membrane
During the intraerythrocytic asexual cycle malaria parasites acquire nutrients and other solutes thr...
Intraerythrocytic growth of the human malaria parasite Plasmodium falciparum depends on delivery of ...
The plasmodial surface anion channel mediates uptake of nutrients and other solutes into erythrocyte...
The human malaria parasite Plasmodium falciparum increases red blood cell membrane permeability duri...
SummaryDevelopment of malaria parasites within vertebrate erythrocytes requires nutrient uptake at t...
ABSTRACT The plasmodial surface anion channel (PSAC) increases erythrocyte permeability to many solu...
ABSTRACT Malaria parasites increase host erythrocyte permeability to ions and nutrients via a broad-...
AbstractErythrocytes infected with malaria parasites have increased permeability to various solutes....
The plasmodial surface anion channel (PSAC) increases eryth-rocyte permeability to many solutes in m...
Malaria parasites activate a broad-selectivity ion channel on their host erythrocyte membrane to obt...
AbstractThe properties of the malaria parasite-induced permeability pathways in the host red blood c...
The malaria parasite, Plasmodium falciparum, requires large amounts of nutrients to sustain its rapi...
AbstractWe recently identified a voltage-dependent anion channel on the surface of human red blood c...
Current malaria research is geared toward identifying novel targets for malaria chemotherapy because...
Erythrocytes infected with malaria parasites have increased permeability to ions and nutrients, as m...
During the intraerythrocytic asexual cycle malaria parasites acquire nutrients and other solutes thr...
Intraerythrocytic growth of the human malaria parasite Plasmodium falciparum depends on delivery of ...
The plasmodial surface anion channel mediates uptake of nutrients and other solutes into erythrocyte...
The human malaria parasite Plasmodium falciparum increases red blood cell membrane permeability duri...
SummaryDevelopment of malaria parasites within vertebrate erythrocytes requires nutrient uptake at t...
ABSTRACT The plasmodial surface anion channel (PSAC) increases erythrocyte permeability to many solu...
ABSTRACT Malaria parasites increase host erythrocyte permeability to ions and nutrients via a broad-...
AbstractErythrocytes infected with malaria parasites have increased permeability to various solutes....
The plasmodial surface anion channel (PSAC) increases eryth-rocyte permeability to many solutes in m...
Malaria parasites activate a broad-selectivity ion channel on their host erythrocyte membrane to obt...
AbstractThe properties of the malaria parasite-induced permeability pathways in the host red blood c...
The malaria parasite, Plasmodium falciparum, requires large amounts of nutrients to sustain its rapi...
AbstractWe recently identified a voltage-dependent anion channel on the surface of human red blood c...
Current malaria research is geared toward identifying novel targets for malaria chemotherapy because...
Erythrocytes infected with malaria parasites have increased permeability to ions and nutrients, as m...
During the intraerythrocytic asexual cycle malaria parasites acquire nutrients and other solutes thr...
Intraerythrocytic growth of the human malaria parasite Plasmodium falciparum depends on delivery of ...
The plasmodial surface anion channel mediates uptake of nutrients and other solutes into erythrocyte...