Necroptosis has emerged as an important facet of both host defense and inflammatory disease. In this issue, Rodrigue-Gervais et al. (2014) demonstrate a role for the cell death regulator cIAP2 in maintaining lung homeostasis during influenza infection. Loss of cIAP2 promotes necroptosis of lung tissues, leading to host death
SummaryPathogen and danger recognition by the inflammasome activates inflammatory caspases that medi...
A key host innate immune response to pathogens is the rapid activation of "inflammasomes". The infla...
Host cells infected with obligate intracellular bacteria Chlamydia trachomatis are profoundly resist...
Necroptosis has emerged as an important facet of both host defense and inflammatory disease. In this...
SummaryCellular inhibitors of apoptosis proteins (cIAPs) are essential regulators of cell death and ...
SummaryCellular inhibitors of apoptosis proteins (cIAPs) are essential regulators of cell death and ...
Background. cIAP2 is involved in necroptosis as a key upstream regulation factor. We aimed to invest...
SummaryPathogen and danger recognition by the inflammasome activates inflammatory caspases that medi...
The cellular inhibitor of apoptosis 2 (cIAP2/HIAP1) is a potent inhibitor of apoptotic death. In con...
Eukaryotic cells can die from physical trauma, resulting in necrosis. Alternately, they can die via ...
Cellular inhibitor of apoptosis proteins (cIAPs) have emerged as important anti-cell death mediators...
Eukaryotic cells can die from physical trauma, resulting in necrosis. Alternately, they can die via ...
Cell death is an effective strategy to limit intracellular infections. Canonical inflammasomes, incl...
Cell death is an effective strategy to limit intracellular infections. Canonical inflammasomes, incl...
AbstractAutophagy, a stress response activated in influenza A virus infection helps the cell avoid a...
SummaryPathogen and danger recognition by the inflammasome activates inflammatory caspases that medi...
A key host innate immune response to pathogens is the rapid activation of "inflammasomes". The infla...
Host cells infected with obligate intracellular bacteria Chlamydia trachomatis are profoundly resist...
Necroptosis has emerged as an important facet of both host defense and inflammatory disease. In this...
SummaryCellular inhibitors of apoptosis proteins (cIAPs) are essential regulators of cell death and ...
SummaryCellular inhibitors of apoptosis proteins (cIAPs) are essential regulators of cell death and ...
Background. cIAP2 is involved in necroptosis as a key upstream regulation factor. We aimed to invest...
SummaryPathogen and danger recognition by the inflammasome activates inflammatory caspases that medi...
The cellular inhibitor of apoptosis 2 (cIAP2/HIAP1) is a potent inhibitor of apoptotic death. In con...
Eukaryotic cells can die from physical trauma, resulting in necrosis. Alternately, they can die via ...
Cellular inhibitor of apoptosis proteins (cIAPs) have emerged as important anti-cell death mediators...
Eukaryotic cells can die from physical trauma, resulting in necrosis. Alternately, they can die via ...
Cell death is an effective strategy to limit intracellular infections. Canonical inflammasomes, incl...
Cell death is an effective strategy to limit intracellular infections. Canonical inflammasomes, incl...
AbstractAutophagy, a stress response activated in influenza A virus infection helps the cell avoid a...
SummaryPathogen and danger recognition by the inflammasome activates inflammatory caspases that medi...
A key host innate immune response to pathogens is the rapid activation of "inflammasomes". The infla...
Host cells infected with obligate intracellular bacteria Chlamydia trachomatis are profoundly resist...
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