Add to ORKGWe hypothesized that repetitive umbilical cord occlusions (UCOs) leading to severe acidemia will stimulate a placental and fetal inflammatory response associated with brain injury, which will be exacerbated by chronic hypoxemia and low-grade infection. Chronically instrumented fetal sheep served as controls or underwent repetitive UCOs for up to 4 hours or until fetal arterial pH was2saturation pre-UCOs of \u3e55% an
Fetal hypoxia contributes significantly to the pathogenesis of permanent perinatal brain injury. Cer...
Hypoxia can significantly contribute to the development of permanent brain injury in the term neonat...
Background: Fetal hypoxia contributes significantly to the pathogenesis of permanent perinatal brain...
Fetal hypoxic episodes may occur antepartum with the potential to induce systemic and cerebral infla...
Department of Obstetrics and Gynecology, University Hospital Maastricht, The Netherlands. OBJECTIVE:...
Chorioamnionitis and fetal sepsis can induce a fetal inflammatory response syndrome (FIRS) which is ...
Objective: to determine whether repetitive umbilical cord occlusion resulting in fetal hypoxemia but...
Background: Infants born preterm following exposure to in utero inflammation/chorioamnionitis are at...
Fetal brain injury is a major cause for many long-term neurodevelopmental sequelae after birth.My th...
\u3cp\u3eBackground: Hypoxic-ischemic encephalopathy (HIE) is one of the most important causes of br...
OBJECTIVE: Intrauterine infection has been linked to brain injury in human infants, although the mec...
INTRODUCTION: Preterm birth is a major cause of infant morbidity and long-term disability, and is as...
Fetal growth and development are controlled by genetic and environmental factors. There is overwhelm...
This study aimed to determine the effect of varying degrees of intermittent umbilical cord occlusion...
The purpose of this study was to compare the regional distribution of apoptotic cells in the near te...
Fetal hypoxia contributes significantly to the pathogenesis of permanent perinatal brain injury. Cer...
Hypoxia can significantly contribute to the development of permanent brain injury in the term neonat...
Background: Fetal hypoxia contributes significantly to the pathogenesis of permanent perinatal brain...
Fetal hypoxic episodes may occur antepartum with the potential to induce systemic and cerebral infla...
Department of Obstetrics and Gynecology, University Hospital Maastricht, The Netherlands. OBJECTIVE:...
Chorioamnionitis and fetal sepsis can induce a fetal inflammatory response syndrome (FIRS) which is ...
Objective: to determine whether repetitive umbilical cord occlusion resulting in fetal hypoxemia but...
Background: Infants born preterm following exposure to in utero inflammation/chorioamnionitis are at...
Fetal brain injury is a major cause for many long-term neurodevelopmental sequelae after birth.My th...
\u3cp\u3eBackground: Hypoxic-ischemic encephalopathy (HIE) is one of the most important causes of br...
OBJECTIVE: Intrauterine infection has been linked to brain injury in human infants, although the mec...
INTRODUCTION: Preterm birth is a major cause of infant morbidity and long-term disability, and is as...
Fetal growth and development are controlled by genetic and environmental factors. There is overwhelm...
This study aimed to determine the effect of varying degrees of intermittent umbilical cord occlusion...
The purpose of this study was to compare the regional distribution of apoptotic cells in the near te...
Fetal hypoxia contributes significantly to the pathogenesis of permanent perinatal brain injury. Cer...
Hypoxia can significantly contribute to the development of permanent brain injury in the term neonat...
Background: Fetal hypoxia contributes significantly to the pathogenesis of permanent perinatal brain...