Add to ORKGIn this issue of Developmental Cell, Waldron et al. (2016) identify an interaction between a master regulator of heart development, TBX5, and the NuRD complex and describe how mutations affecting the interaction may contribute to congenital heart disease. Furthermore, these interactions may have contributed to the evolution of cardiac septation
TBX5 is a member of the T-box transcription factor family and is primarily known for its role in car...
AbstractDominant mutations in the T-box transcription factor gene TBX5 cause Holt–Oram syndrome (HOS...
Transcription factors (TFs) are thought to function with partners to achieve specificity and precise...
SummaryHuman mutations in the cardiac transcription factor gene TBX5 cause congenital heart disease ...
Human mutations in the cardiac transcription factor gene TBX5 cause Congenital Heart Disease (CHD), ...
SummaryHuman mutations in the cardiac transcription factor gene TBX5 cause congenital heart disease ...
In humans, septal defects are among the most prevalent congenital heart diseases, but their cellular...
In this issue of Developmental Cell, Waldron et al. (2016) identify an interaction between a master ...
SummaryThe developmental mechanisms underlying human congenital heart disease (CHD) are poorly under...
Congenital heart disease (CHD) remains the most common congenital malformation, with defects in card...
<div><p>Mutations of the <em>Wnt5a</em> gene, encoding a ligand of the non-canonical Wnt pathway, an...
Mutations of the Wnt5a gene, encoding a ligand of the non-canonical Wnt pathway, and the Ror2 gene, ...
Mutations of the Wnt5a gene, encoding a ligand of the non-canonical Wnt pathway, and the Ror2 gene, ...
Mutations of the Wnt5a gene, encoding a ligand of the non-canonical Wnt pathway, and the Ror2 gene, ...
Mutations of the Wnt5a gene, encoding a ligand of the non-canonical Wnt pathway, and the Ror2 gene, ...
TBX5 is a member of the T-box transcription factor family and is primarily known for its role in car...
AbstractDominant mutations in the T-box transcription factor gene TBX5 cause Holt–Oram syndrome (HOS...
Transcription factors (TFs) are thought to function with partners to achieve specificity and precise...
SummaryHuman mutations in the cardiac transcription factor gene TBX5 cause congenital heart disease ...
Human mutations in the cardiac transcription factor gene TBX5 cause Congenital Heart Disease (CHD), ...
SummaryHuman mutations in the cardiac transcription factor gene TBX5 cause congenital heart disease ...
In humans, septal defects are among the most prevalent congenital heart diseases, but their cellular...
In this issue of Developmental Cell, Waldron et al. (2016) identify an interaction between a master ...
SummaryThe developmental mechanisms underlying human congenital heart disease (CHD) are poorly under...
Congenital heart disease (CHD) remains the most common congenital malformation, with defects in card...
<div><p>Mutations of the <em>Wnt5a</em> gene, encoding a ligand of the non-canonical Wnt pathway, an...
Mutations of the Wnt5a gene, encoding a ligand of the non-canonical Wnt pathway, and the Ror2 gene, ...
Mutations of the Wnt5a gene, encoding a ligand of the non-canonical Wnt pathway, and the Ror2 gene, ...
Mutations of the Wnt5a gene, encoding a ligand of the non-canonical Wnt pathway, and the Ror2 gene, ...
Mutations of the Wnt5a gene, encoding a ligand of the non-canonical Wnt pathway, and the Ror2 gene, ...
TBX5 is a member of the T-box transcription factor family and is primarily known for its role in car...
AbstractDominant mutations in the T-box transcription factor gene TBX5 cause Holt–Oram syndrome (HOS...
Transcription factors (TFs) are thought to function with partners to achieve specificity and precise...