Common genetic loci influencing plasma homocysteine concentrations and their effect on risk of coronary artery disease.
- van Meurs, JB
- Pare, G
- Schwartz, SM
- Hazra, A
- Tanaka, T
- Vermeulen, SH
- Cotlarciuc, I
- Yuan, X
- Mälarstig, A
- Bandinelli, S
- Bis, JC
- Blom, H
- Brown, MJ
- Chen, C
- Chen, YD
- Clarke, RJ
- Dehghan, A
- Erdmann, J
- Ferrucci, L
- Hamsten, A
- Hofman, A
- Hunter, DJ
- Goel, A
- Johnson, AD
- Kathiresan, S
- Kampman, E
- Kiel, DP
- Kiemeney, LA
- Chambers, JC
- Kraft, P
- Lindemans, J
- McKnight, B
- Nelson, CP
- O'Donnell, CJ
- Psaty, BM
- Ridker, PM
- Rivadeneira, F
- Rose, LM
- Seedorf, U
- Siscovick, DS
- Schunkert, H
- Selhub, J
- Ueland, PM
- Vollenweider, P
- Waeber, G
- Waterworth, DM
- Watkins, H
- Witteman, JC
- den Heijer, M
- Jacques, P
- Uitterlinden, AG
- Kooner, JS
- Rader, DJ
- Reilly, MP
- Mooser, V
- Chasman, DI
- Samani, NJ
- Ahmadi, KR
Publication date
January 2020
DOI Publisher
American Society for Nutrition Journal
American Journal of Clinical NutritionAbstract
The strong observational association between total homocysteine (tHcy) concentrations and risk of coronary artery disease (CAD) and the null associations in the homocysteine-lowering trials have prompted the need to identify genetic variants associated with homocysteine concentrations and risk of CAD
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