After HSV infection, some trigeminal ganglion neurons support productive cycle gene expression, while in other neurons the virus establishes a latent infection. We previously demonstrated that HSV-1 and HSV-2 preferentially establish latent infection in A
AbstractThe presence of wild-type herpes simplex virus type 1 (HSV-1) and several latency associated...
Although recurrent Herpes simplex virus type 1 (HSV-1) infections are quite common in humans, little...
Permissive herpes simplex virus (HSV) infection in tissue culture results in host cell destruction. ...
After HSV infection, some trigeminal ganglion neurons support productive cycle gene expression, whil...
Herpes simplex virus (HSV) persists in the human population by establishing long-term latent infecti...
Herpes simplex virus 1 (HSV-1) and HSV-2 establish latency in different neuronal subtypes (A5+ and K...
The herpes simplex virus type 1 (HSV-1) mutant in1814 lacks the ability to trans-activate immediate...
The persistence of herpes simplex virus (HSV) and the diseases that it causes in the human populatio...
Following primary infection Herpes simplex virus-1 (HSV-1) establishes lifelong latency in the neuro...
Following primary infection Herpes simplex virus-1 (HSV-1) establishes lifelong latency in the neuro...
Herpes simplex virus 1 (HSV-1) can establish lifelong latency in human trigeminal ganglia. Latently ...
Following productive, lytic infection in epithelia, herpes simplex virus type 1 (HSV-1) establishes ...
AbstractWe performed in situ hybridization to determine the cell type specific accumulation of the i...
Following primary infection Herpes simplex virus-1 (HSV-1) establishes lifelong latency in the neuro...
Herpes simplex virus (HSV) replicates in peripheral tissues and forms latent infections in neurons o...
AbstractThe presence of wild-type herpes simplex virus type 1 (HSV-1) and several latency associated...
Although recurrent Herpes simplex virus type 1 (HSV-1) infections are quite common in humans, little...
Permissive herpes simplex virus (HSV) infection in tissue culture results in host cell destruction. ...
After HSV infection, some trigeminal ganglion neurons support productive cycle gene expression, whil...
Herpes simplex virus (HSV) persists in the human population by establishing long-term latent infecti...
Herpes simplex virus 1 (HSV-1) and HSV-2 establish latency in different neuronal subtypes (A5+ and K...
The herpes simplex virus type 1 (HSV-1) mutant in1814 lacks the ability to trans-activate immediate...
The persistence of herpes simplex virus (HSV) and the diseases that it causes in the human populatio...
Following primary infection Herpes simplex virus-1 (HSV-1) establishes lifelong latency in the neuro...
Following primary infection Herpes simplex virus-1 (HSV-1) establishes lifelong latency in the neuro...
Herpes simplex virus 1 (HSV-1) can establish lifelong latency in human trigeminal ganglia. Latently ...
Following productive, lytic infection in epithelia, herpes simplex virus type 1 (HSV-1) establishes ...
AbstractWe performed in situ hybridization to determine the cell type specific accumulation of the i...
Following primary infection Herpes simplex virus-1 (HSV-1) establishes lifelong latency in the neuro...
Herpes simplex virus (HSV) replicates in peripheral tissues and forms latent infections in neurons o...
AbstractThe presence of wild-type herpes simplex virus type 1 (HSV-1) and several latency associated...
Although recurrent Herpes simplex virus type 1 (HSV-1) infections are quite common in humans, little...
Permissive herpes simplex virus (HSV) infection in tissue culture results in host cell destruction. ...