N-Terminal Truncated Intracellular Matrix Metalloproteinase-2 Induces Cardiomyocyte Hypertrophy, Inflammation and Systolic Heart Failure

Raffai, Robert
Karliner, Joel
Lovett, David
Lovett, DH
Mahimkar, R
Raffai, RL
Cape, L
Zhu, BQ
Jin, ZQ
Baker, AJ
Karliner, JS

Publication date

July 2013

Publisher

eScholarship, University of California

Abstract

Matrix metalloproteinase-2 (MMP-2) is increasingly recognized as a major contributor to progressive cardiac injury within the setting of ischemia-reperfusion injury and ischemic ventricular remodeling. A common feature of these conditions is an increase i

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N-Terminal Truncated Intracellular Matrix Metalloproteinase-2 Induces Cardiomyocyte Hypertrophy, Inflammation and Systolic Heart Failure

Abstract

Extracted data

N-Terminal Truncated Intracellular Matrix Metalloproteinase-2 Induces Cardiomyocyte Hypertrophy, Inflammation and Systolic Heart Failure

Abstract

Extracted data

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