Research into molecular apoptosis mechanisms in patients with cardio-vascular diseases is one of the pressing issues in modern medicine. Many mechanisms of this phenomenon still remain unexamined, the role of inductors and regulators in cardiomyocyte apoptosis has not yet been fully explained. This review summarizes research data that analyze the role that various groups of cell death inductors and regulators, such as FasL, Fas-R and mitochondrial apoptosis factors. Influence of the Bcl-2 protein family as apoptosis regulators is described. Developments in programmed cell death are reviewed.Key words: apoptosis, cell death mechanisms, mitochondria, cardiamyocytes
Programmed cell death, apoptosis, plays an integral role in a variety of biological events, e.g. mor...
Apoptosis or programmed cell death is an essential physiological process that plays a critical role ...
Apoptosis or programmed cell death is genetically determined process to destroy cells for the mainta...
Research into molecular apoptosis mechanisms in patients with cardio-vascular diseases is one of the...
Cardiovascular disease is a leading cause of death worldwide. Loss of function or death of cardiomyo...
Cardiovascular disease is a leading cause of death worldwide. Loss of function or death of cardiomyo...
Cardiovascular disease is a leading cause of death worldwide. In recent years it has emerged that lo...
Cardiovascular disease is a leading cause of death worldwide. In recent years it has emerged that lo...
Cardiac myocyte death, whether through necrotic or apoptotic mechanisms, is a contributing factor to...
A defining cellular event in the transition from compensated hypertrophy to dilated cardiomyopathy i...
PubMed ID: 12460831Apoptosis is defined as a genetically programmed cell death. Apoptotic cell death...
Apoptosis, or programmed cell death, is a process through which multicellular organisms dis-pose of ...
In recent years, apoptosis has increasingly drawn the attention of both oncologists and cardiologist...
In recent years, apoptosis has increasingly drawn the attention of both oncologists and cardiologist...
The leading cause of death in developed countries is cardiovascular disease, where coronary heart di...
Programmed cell death, apoptosis, plays an integral role in a variety of biological events, e.g. mor...
Apoptosis or programmed cell death is an essential physiological process that plays a critical role ...
Apoptosis or programmed cell death is genetically determined process to destroy cells for the mainta...
Research into molecular apoptosis mechanisms in patients with cardio-vascular diseases is one of the...
Cardiovascular disease is a leading cause of death worldwide. Loss of function or death of cardiomyo...
Cardiovascular disease is a leading cause of death worldwide. Loss of function or death of cardiomyo...
Cardiovascular disease is a leading cause of death worldwide. In recent years it has emerged that lo...
Cardiovascular disease is a leading cause of death worldwide. In recent years it has emerged that lo...
Cardiac myocyte death, whether through necrotic or apoptotic mechanisms, is a contributing factor to...
A defining cellular event in the transition from compensated hypertrophy to dilated cardiomyopathy i...
PubMed ID: 12460831Apoptosis is defined as a genetically programmed cell death. Apoptotic cell death...
Apoptosis, or programmed cell death, is a process through which multicellular organisms dis-pose of ...
In recent years, apoptosis has increasingly drawn the attention of both oncologists and cardiologist...
In recent years, apoptosis has increasingly drawn the attention of both oncologists and cardiologist...
The leading cause of death in developed countries is cardiovascular disease, where coronary heart di...
Programmed cell death, apoptosis, plays an integral role in a variety of biological events, e.g. mor...
Apoptosis or programmed cell death is an essential physiological process that plays a critical role ...
Apoptosis or programmed cell death is genetically determined process to destroy cells for the mainta...