[No abstract available]7081523Lin, C.L., Wang, F.S., Kuo, Y.R., Ras modulation of superoxide activates ERK-dependent fibronectin expression in diabetes-induced renal injuries (2006) Kidney Int, 69, pp. 1593-1600Schiekofer, S., Andrassy, M., Chen, J., Acute hyperglycemia causes intracellular formation of CML and activation of ras, p42/44 MAPK, and nuclear factorkB in PBMCs (2003) Diabetes, 52, pp. 621-63
grantor: University of TorontoHigh glucose causes diacylglycerol (DAG) accumulation and pr...
High ambient glucose exerts its injurious effects on renal cells through nonenzymatic and enzymatic ...
The pathogenesis of diabetic nephropathy (DN) remains incompletely understood. In previous studies, ...
Although previous studies have demonstrated that diabetic nephropathy is attributable to early extra...
Reactive oxygen species as glucose signaling molecules in mesangial cells cultured under high glucos...
Abnormal glycemia is frequently along with nephritis, whose pathogenesis is unexplicit. Here, we inv...
High glucose evokes an intrinsic proapoptotic signaling pathway in mesangial cells.BackgroundIn resp...
Amino acids injure mesangial cells by advanced glycation end products, oxidative stress, and protein...
Role of oxidative stress in advanced glycation end product-induced mesangial cell activation.Backgro...
Hyperglycemia and hyperlipidemia are considered critical to the development of diabetic nephropathy....
This study investigated the role of advanced glycation end products (AGEs) in mediating protein kina...
Background : The p38 mitogen-activated protein kinase (MAPK) pathway is activated by several stress ...
grantor: University of TorontoIn diabetes, glomerular mesangial cells (MCs) are transforme...
Diabetes induces the onset and progression of renal injury through causing hemodynamic dysregulation...
AIMS/HYPOTHESIS: Infiltration of mononuclear cells and glomerular enlargement accompanied by glome...
grantor: University of TorontoHigh glucose causes diacylglycerol (DAG) accumulation and pr...
High ambient glucose exerts its injurious effects on renal cells through nonenzymatic and enzymatic ...
The pathogenesis of diabetic nephropathy (DN) remains incompletely understood. In previous studies, ...
Although previous studies have demonstrated that diabetic nephropathy is attributable to early extra...
Reactive oxygen species as glucose signaling molecules in mesangial cells cultured under high glucos...
Abnormal glycemia is frequently along with nephritis, whose pathogenesis is unexplicit. Here, we inv...
High glucose evokes an intrinsic proapoptotic signaling pathway in mesangial cells.BackgroundIn resp...
Amino acids injure mesangial cells by advanced glycation end products, oxidative stress, and protein...
Role of oxidative stress in advanced glycation end product-induced mesangial cell activation.Backgro...
Hyperglycemia and hyperlipidemia are considered critical to the development of diabetic nephropathy....
This study investigated the role of advanced glycation end products (AGEs) in mediating protein kina...
Background : The p38 mitogen-activated protein kinase (MAPK) pathway is activated by several stress ...
grantor: University of TorontoIn diabetes, glomerular mesangial cells (MCs) are transforme...
Diabetes induces the onset and progression of renal injury through causing hemodynamic dysregulation...
AIMS/HYPOTHESIS: Infiltration of mononuclear cells and glomerular enlargement accompanied by glome...
grantor: University of TorontoHigh glucose causes diacylglycerol (DAG) accumulation and pr...
High ambient glucose exerts its injurious effects on renal cells through nonenzymatic and enzymatic ...
The pathogenesis of diabetic nephropathy (DN) remains incompletely understood. In previous studies, ...