Inheritance of Aspergillus Ear Rot and Aflatoxin Resistance From the Tex6 Corn Line

56 p.Thesis (Ph.D.)--University of Illinois at Urbana-Champaign, 1998.F$\sb1$ means deviated from the midparent value toward resistance for aflatoxin production and toward susceptibility for ear rot in both crosses. In the B73 $\times$ Tex6 cross, generation mean analysis indicated additive gene action was the most important type of gene action controlling resistance to ear rot and to aflatoxin production. Mo17 was less susceptible to both traits, so the resistance from Tex6 in crosses with Mo17 was not well defined. Broad-sense heritabilities were 58% for ear rot and 63% for aflatoxin production in the Mo17 $\times$ Tex6 cross, and 66% for ear rot and 65% for aflatoxin production in the B73 $\times$ Tex6 cross. Narrow-sense heritabilities were 39% for ear rot and 45% for aflatoxin production in the B73 $\times$ Tex6 cross. Expected gain from F$\sb3$ family selection was 11.1% for ear rot and 1.5% for aflatoxin production. Selection will be more effective within F$\sb3$ families of the B73 $\times$ Tex6 cross than F$\sb3$ families of the Mo17 $\times$ Tex6 cross, particularly in environments favorable for disease development.U of I OnlyRestricted to the U of I community idenfinitely during batch ingest of legacy ETD