Add to ORKGCardiomyocyte apoptosis and hypertrophy have been studied in the left and right ventricular myocardium of spontaneously hypertensive rats without treatment and after 10-day administration of the ETA-receptor antagonist BQ-123. It is established that BQ-123 prevents the activation of cardiomyocyte apoptosis and significantly decreases the extent of hypertrophy development in the left ventricular myocardium, but does not influence the same mechanisms in the right ventricular myocardium
<p>(A) and (B) CHOP was increased in AAC rats (4 or 8 weeks after operation), and metoprolol (30 mg/...
Previous findings have shown that hypotensive doses of losartan prevent the excess of apoptosis pres...
Background Previous studies have demonstrated that an-giotensin II (Ang II) acts as a growth-promoti...
Cardiomyocyte apoptosis and hypertrophy have been studied in the left and right ventricular myocardi...
This study was performed to evaluate the effects on mechanical and metabolic function of endogenous ...
We aimed to investigate the effect of etanercept, a tumor necrosis factor-α (TNF-α) inhibitor, on ra...
The objective of the present study was to investigate the possible impact of specific endothelin-A (...
Myocardial cell apoptosis in genetically determined arterial hypertension was investigated. Male spo...
1. 1. The cardiovascular effects of SRT6b in control and BQ-123, a specific ETA receptor antagonist,...
<p>. (A) to (C) showed β-AR blockers attenuated cardiac hypertrophy in rats induced by abdominal aor...
Increased apoptosis has been reported in the heart of rats with spontaneous hypertension and cardiac...
Background: Apoptosis plays a major role in the transition to heart failure (HF) in systemic hyperte...
We previously reported that the AT1 receptor antagonist valsartan and the angiotensin converting enz...
In this study, we evaluated whether blocking myeloid differentiation factor-88 (MyD88) could decreas...
Objectives: The aim of the current study was to address the issue of cardiomyocyte apoptosis as a po...
<p>(A) and (B) CHOP was increased in AAC rats (4 or 8 weeks after operation), and metoprolol (30 mg/...
Previous findings have shown that hypotensive doses of losartan prevent the excess of apoptosis pres...
Background Previous studies have demonstrated that an-giotensin II (Ang II) acts as a growth-promoti...
Cardiomyocyte apoptosis and hypertrophy have been studied in the left and right ventricular myocardi...
This study was performed to evaluate the effects on mechanical and metabolic function of endogenous ...
We aimed to investigate the effect of etanercept, a tumor necrosis factor-α (TNF-α) inhibitor, on ra...
The objective of the present study was to investigate the possible impact of specific endothelin-A (...
Myocardial cell apoptosis in genetically determined arterial hypertension was investigated. Male spo...
1. 1. The cardiovascular effects of SRT6b in control and BQ-123, a specific ETA receptor antagonist,...
<p>. (A) to (C) showed β-AR blockers attenuated cardiac hypertrophy in rats induced by abdominal aor...
Increased apoptosis has been reported in the heart of rats with spontaneous hypertension and cardiac...
Background: Apoptosis plays a major role in the transition to heart failure (HF) in systemic hyperte...
We previously reported that the AT1 receptor antagonist valsartan and the angiotensin converting enz...
In this study, we evaluated whether blocking myeloid differentiation factor-88 (MyD88) could decreas...
Objectives: The aim of the current study was to address the issue of cardiomyocyte apoptosis as a po...
<p>(A) and (B) CHOP was increased in AAC rats (4 or 8 weeks after operation), and metoprolol (30 mg/...
Previous findings have shown that hypotensive doses of losartan prevent the excess of apoptosis pres...
Background Previous studies have demonstrated that an-giotensin II (Ang II) acts as a growth-promoti...