Rapidly expanding evidence increasingly strengthens the evidence linking psychological factors to asthma and allergy expression. Parallel studies in animals and humans demonstrating the influence of prenatal maternal stress and early caregiving experiences on the disrupted regulation of defensive biological systems [eg, sympathetic and adrenomedullary (SAM) system and the hypothalamicpituitary-adrenocortical (HPA) axis] provide strong proof of concept for this line of research. The consequent altered neuroimmune responses may influence the expression of immune-mediated disorders such as asthma as well as enhance an individual's susceptibility to other environmental factors that may also contribute to asthma risk
In developing youth, allergic asthma is the most common chronic condition, with 9%–10% of youth affe...
The hygiene hypothesis states that childhood asthma develops as a result of decreased exposure to in...
<b><i>Background:</i></b> Bronchial asthma is characterized by type 2 T helper (Th2) cell inflammati...
Adverse childhood experiences such as abuse and neglect are associated with subsequent immune dysreg...
Allergic manifestations are increasingly common in infants and children. Accumulating evidence sugge...
Little is known about how prenatal maternal stress (PNMS) influences risks of asthma in humans. In t...
[Truncated abstract] Introduction The western world has seen a dramatic increase in the prevalence o...
<b>BACKGROUND:</b> High levels of stress have been shown to predict the onset of asthma ...
A common explanation for the origins and rising prevalence of asthma is that they involve complex in...
UnrestrictedAsthma is the most common chronic inflammatory disease in childhood. The disease often s...
BACKGROUND: Prenatal maternal stress may influence offspring's atopic risk through sustained cortiso...
Although psychosocial stress has been linked to clinical asthma outcomes, controlled, laboratory par...
UnrestrictedChildhood asthma prevalence is often higher in areas of low socioeconomic status (SES), ...
BACKGROUND: Recent findings suggest that the natural history of asthma and atopy begins in foetal l...
ABSTRACTBackgroundPsychological stress has long been recognized to be associated with asthma symptom...
In developing youth, allergic asthma is the most common chronic condition, with 9%–10% of youth affe...
The hygiene hypothesis states that childhood asthma develops as a result of decreased exposure to in...
<b><i>Background:</i></b> Bronchial asthma is characterized by type 2 T helper (Th2) cell inflammati...
Adverse childhood experiences such as abuse and neglect are associated with subsequent immune dysreg...
Allergic manifestations are increasingly common in infants and children. Accumulating evidence sugge...
Little is known about how prenatal maternal stress (PNMS) influences risks of asthma in humans. In t...
[Truncated abstract] Introduction The western world has seen a dramatic increase in the prevalence o...
<b>BACKGROUND:</b> High levels of stress have been shown to predict the onset of asthma ...
A common explanation for the origins and rising prevalence of asthma is that they involve complex in...
UnrestrictedAsthma is the most common chronic inflammatory disease in childhood. The disease often s...
BACKGROUND: Prenatal maternal stress may influence offspring's atopic risk through sustained cortiso...
Although psychosocial stress has been linked to clinical asthma outcomes, controlled, laboratory par...
UnrestrictedChildhood asthma prevalence is often higher in areas of low socioeconomic status (SES), ...
BACKGROUND: Recent findings suggest that the natural history of asthma and atopy begins in foetal l...
ABSTRACTBackgroundPsychological stress has long been recognized to be associated with asthma symptom...
In developing youth, allergic asthma is the most common chronic condition, with 9%–10% of youth affe...
The hygiene hypothesis states that childhood asthma develops as a result of decreased exposure to in...
<b><i>Background:</i></b> Bronchial asthma is characterized by type 2 T helper (Th2) cell inflammati...