Biological and Sanitary Effects of the Exposures to Non Ionizing Electromagnetic Fields (EMF) and the MUOS Case

Recent and very recent scientific literature shows that both biological and sanitary effects of EMF radiations – from the extremely low frequency magnetic fields (ELF/EMF) to the high and very high radiofrequencies (RF/EMF) – are clearly established and occur even at very low exposure levels. Overall, there are now almost 4.000 experimental studies that report a variety of short and medium-term effects of EMF, which support the biological plausibility of the increased risks of their long-term genotoxic, carcinogenic and neurodegenerative consequences on exposed human populations. For instance, EMF exposures of cultured mammaliant cells, experimental animals and human subjects may induce genetic and epigenetic effects, such as single and double strand DNA damages, chromosomal aberrations, micronuclei, sister-chromatid exchanges, alteration or loss of the DNA damage repair processes, abnormal DNA transcription and protein functions, etc.; stimulation of heat-shock protein synthesis; inhibition of apoptosis (programmed cell death); damages to cellular macromolecules due to the impairment of the inactivation of free radicals and the consequent oxidative stress on account of the inhibition of melatonin syntesis and the stimulation of the Fenton’s reaction; modification of the cell membrane permeability and the consequent alteration of the flow of biologically important ions such as Calcium; alteration of the function of the immune system; serious impacts on sperm morphology and functional with consequent effects on the offspring; alterations of the brain functions as a consequence of the interference of a EMF on cerebral frequencies, etc. Many of these bioeffects can reasonably be presumed to result in adverse health effects if the exposures are prolonged or chronic. This is because they interfere with normal body processes (disrupt homeostasis), prevent the body from healing damaged DNA, produce immune system imbalances, metabolic disruption and lower resilience to disease across multiple pathways