cTBS over contralesional homologue areas deteriorates speech output in isolated apraxia of speech after stroke

Apraxia of speech (AOS) is an impairment of motor speech planning/programming, characterized by a combination of phonemic segmental changes and articulatory distortions [1]. AOS is most often accompanied by aphasia, whereas isolated forms may rarely occur after focal damage to the left precentral gyrus [1]. How AOS recovers after brain damage is poorly understood [2]. In particular, it is not known whether recovery of AOS solely depends on the functional reorganisation of perilesional areas [3,4], or whether it also depends on the compensation through contralesional homologue areas