AbstractIn a neuron's dendritic spine, the location of CaMKII is controlled by a number of interacting factors, including its ability to bind calcium/calmodulin, its phosphorylation state and the synthesis of new subunits in the dendrites.New studies have shown that the exact location of CaMKII is crucial for the form and endurance of synaptic plasticity
Calcium-calmodulin (CaM)-dependent protein kinase II (CaMKII) is the most abundant protein in excita...
AbstractNeurite extension and branching are important neuronal plasticity mechanisms that can lead t...
Ca2+/calmodulin-dependent protein kinase II (CaMKII) is essential for long-term potentiation (LTP) o...
In a neuron's dendritic spine, the location of CaMKII is controlled by a number of interacting facto...
In a neuron's dendritic spine, the location of CaMKII is controlled by a number of interacting facto...
In a neuron's dendritic spine, the location of CaMKII is controlled by a number of interacting facto...
AbstractIn a neuron's dendritic spine, the location of CaMKII is controlled by a number of interacti...
<p>Synaptic plasticity is the leading candidate for the cellular/molecular basis of learning and mem...
AbstractNeurite extension and branching are important neuronal plasticity mechanisms that can lead t...
SummaryThe structural modification of dendritic spines plays a critical role in synaptic plasticity....
Calcium/calmodulin-dependent protein kinase II (CaM-KII) plays a key role in N-methyl-D-aspartate (N...
Calcium/calmodulin-dependent protein kinase II (CaM-KII) plays a key role in N-methyl-D-aspartate (N...
CaMKII, calcium/calmodulin dependent protein kinase, is an active kinase in the cell that phosphoryl...
Calcium/calmodulin-dependent protein kinase II (CaMKII) plays a key role in N-methyl-D-aspartate (NM...
SummaryCalcium-calmodulin-dependent protein kinase II (CaMKII) is a key mediator of synaptic plastic...
Calcium-calmodulin (CaM)-dependent protein kinase II (CaMKII) is the most abundant protein in excita...
AbstractNeurite extension and branching are important neuronal plasticity mechanisms that can lead t...
Ca2+/calmodulin-dependent protein kinase II (CaMKII) is essential for long-term potentiation (LTP) o...
In a neuron's dendritic spine, the location of CaMKII is controlled by a number of interacting facto...
In a neuron's dendritic spine, the location of CaMKII is controlled by a number of interacting facto...
In a neuron's dendritic spine, the location of CaMKII is controlled by a number of interacting facto...
AbstractIn a neuron's dendritic spine, the location of CaMKII is controlled by a number of interacti...
<p>Synaptic plasticity is the leading candidate for the cellular/molecular basis of learning and mem...
AbstractNeurite extension and branching are important neuronal plasticity mechanisms that can lead t...
SummaryThe structural modification of dendritic spines plays a critical role in synaptic plasticity....
Calcium/calmodulin-dependent protein kinase II (CaM-KII) plays a key role in N-methyl-D-aspartate (N...
Calcium/calmodulin-dependent protein kinase II (CaM-KII) plays a key role in N-methyl-D-aspartate (N...
CaMKII, calcium/calmodulin dependent protein kinase, is an active kinase in the cell that phosphoryl...
Calcium/calmodulin-dependent protein kinase II (CaMKII) plays a key role in N-methyl-D-aspartate (NM...
SummaryCalcium-calmodulin-dependent protein kinase II (CaMKII) is a key mediator of synaptic plastic...
Calcium-calmodulin (CaM)-dependent protein kinase II (CaMKII) is the most abundant protein in excita...
AbstractNeurite extension and branching are important neuronal plasticity mechanisms that can lead t...
Ca2+/calmodulin-dependent protein kinase II (CaMKII) is essential for long-term potentiation (LTP) o...
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