AbstractThe gene responsible for Wiskott–Aldrich syndrome, a disease affecting platelets and lymphocytes, has been cloned and its protein product (WASp) found to interact with the GTPase Cdc42. WASp seems to provide a link between Cdc42 and the actin cytoskeleton, perhaps explaining the cellular defects underlying the disease
Mutations in the gene encoding the Wiskott–Aldrich syndrome protein (WASP) are responsible for Wisko...
AbstractBackground Receptor-mediated signal transduction requires the assembly of multimeric complex...
AbstractThe Wiskott-Aldrich syndrome (WAS) is a human X-linked immunodeficiency resulting from mutat...
AbstractThe Rho family of GTPases control diverse biological processes, including cell morphology an...
The Wiskott-Aldrich Syndrome (WAS) is an × chromosome-linked immunodeficiency disorder. The most com...
The actin cytoskeleton is essential for proper functioning of the immune system by regulat-ing cell ...
The molecular link between the signalling pathway regulating the formation of filopodia and the init...
AbstractBackground: Members of the Rho family of small GTPases play an essential role in controlling...
AbstractRegulation of the actin cytoskeleton is crucial for many aspects of correct and cooperative ...
verrier et al., 2001). Similarly, a knockout mutation of the more ubiquitously expressed N-WASP gene...
AbstractThe Wiskott-Aldrich syndrome (WAS) is a rare immunodeficiency disease affecting mainly plate...
Wiskott-Aldrich syndrome (WAS) is a rare X-linked recessive primary immunodeficiency characterised b...
AbstractWiskott–Aldrich Syndrome (WAS) is caused by mutations in Wiskott-Aldrich Syndrome Protein (W...
AbstractActin polymerization at the cell cortex is thought to provide the driving force for aspects ...
Wiskott Aldrich Syndrome (WAS) is an X-linked recessive disease with clinical symptoms such as throm...
Mutations in the gene encoding the Wiskott–Aldrich syndrome protein (WASP) are responsible for Wisko...
AbstractBackground Receptor-mediated signal transduction requires the assembly of multimeric complex...
AbstractThe Wiskott-Aldrich syndrome (WAS) is a human X-linked immunodeficiency resulting from mutat...
AbstractThe Rho family of GTPases control diverse biological processes, including cell morphology an...
The Wiskott-Aldrich Syndrome (WAS) is an × chromosome-linked immunodeficiency disorder. The most com...
The actin cytoskeleton is essential for proper functioning of the immune system by regulat-ing cell ...
The molecular link between the signalling pathway regulating the formation of filopodia and the init...
AbstractBackground: Members of the Rho family of small GTPases play an essential role in controlling...
AbstractRegulation of the actin cytoskeleton is crucial for many aspects of correct and cooperative ...
verrier et al., 2001). Similarly, a knockout mutation of the more ubiquitously expressed N-WASP gene...
AbstractThe Wiskott-Aldrich syndrome (WAS) is a rare immunodeficiency disease affecting mainly plate...
Wiskott-Aldrich syndrome (WAS) is a rare X-linked recessive primary immunodeficiency characterised b...
AbstractWiskott–Aldrich Syndrome (WAS) is caused by mutations in Wiskott-Aldrich Syndrome Protein (W...
AbstractActin polymerization at the cell cortex is thought to provide the driving force for aspects ...
Wiskott Aldrich Syndrome (WAS) is an X-linked recessive disease with clinical symptoms such as throm...
Mutations in the gene encoding the Wiskott–Aldrich syndrome protein (WASP) are responsible for Wisko...
AbstractBackground Receptor-mediated signal transduction requires the assembly of multimeric complex...
AbstractThe Wiskott-Aldrich syndrome (WAS) is a human X-linked immunodeficiency resulting from mutat...
DOI
Add to ORKG