mutant mice have suggested that GABA function could be required either in the central nervous system or in the palate itself for normal palatogenesis. was sufficient to disrupt palate development. in the central nervous system for normal development of the palate. We suggest that the alterations in GABA signaling lead to non-neural defects such as cleft palate as a secondary effect due to alterations in or elimination of fetal movements
The absence of fragile X mental retardation protein results in the fragile X syndrome (FXS), a commo...
The occurrence of cleft palate in mutant mice offers an opportunity to understand the possible role ...
SUMMARY Apert syndrome is a congenital disorder characterized by severe skull malformations and caus...
Journal ArticleThe functions of neurotransmitters in fetal development are poorly understood. Geneti...
The purpose of this study was to examine the expression of GABAergic proteins in Fmr1 knockout mice ...
Fragile X Syndrome, caused by Fmr1 gene inactivation, is characterized by symptoms including enhance...
Central nervous system GABAergic and glycinergic synaptic activity switches from postsynaptic excita...
Central nervous system GABAergic and glycinergic synaptic activity switches from postsynaptic excita...
Central nervous system GABAergic and glycinergic synaptic activity switches from postsynaptic excita...
Muscle relaxants, anesthetics, and central nervous system depressants administered to pregnant mice ...
Mammalian palate separates the oral and nasal cavities for normal feeding, breathing and speech. The...
Background The β3 subunit of the γ-aminobutyric acid type A receptor (GABAA-R) has been reported to ...
Clefts of the palate are common birth defects requiring extensive treatment. They appear to be cause...
Both glycinergic and GABAergic neurons require the vesicular inhibitory amino acid transporter (VIAA...
Ephs and ephrins are cell surface receptors that bind to each other and initiate distinct, bidirecti...
The absence of fragile X mental retardation protein results in the fragile X syndrome (FXS), a commo...
The occurrence of cleft palate in mutant mice offers an opportunity to understand the possible role ...
SUMMARY Apert syndrome is a congenital disorder characterized by severe skull malformations and caus...
Journal ArticleThe functions of neurotransmitters in fetal development are poorly understood. Geneti...
The purpose of this study was to examine the expression of GABAergic proteins in Fmr1 knockout mice ...
Fragile X Syndrome, caused by Fmr1 gene inactivation, is characterized by symptoms including enhance...
Central nervous system GABAergic and glycinergic synaptic activity switches from postsynaptic excita...
Central nervous system GABAergic and glycinergic synaptic activity switches from postsynaptic excita...
Central nervous system GABAergic and glycinergic synaptic activity switches from postsynaptic excita...
Muscle relaxants, anesthetics, and central nervous system depressants administered to pregnant mice ...
Mammalian palate separates the oral and nasal cavities for normal feeding, breathing and speech. The...
Background The β3 subunit of the γ-aminobutyric acid type A receptor (GABAA-R) has been reported to ...
Clefts of the palate are common birth defects requiring extensive treatment. They appear to be cause...
Both glycinergic and GABAergic neurons require the vesicular inhibitory amino acid transporter (VIAA...
Ephs and ephrins are cell surface receptors that bind to each other and initiate distinct, bidirecti...
The absence of fragile X mental retardation protein results in the fragile X syndrome (FXS), a commo...
The occurrence of cleft palate in mutant mice offers an opportunity to understand the possible role ...
SUMMARY Apert syndrome is a congenital disorder characterized by severe skull malformations and caus...
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