AbstractInflammatory mediators act on peripheral sensory neurons to produce pain and hypersensitivity after tissue injury. In this issue of Neuron, Dina et al. report that inflammatory mediators, such as epinephrine and prostaglandins, appear to couple to specific G protein-coupled receptor signaling pathways through plastic interactions with the cytoskeleton
Copyright © 2015 Roberta Lattanzi et al. This is an open access article distributed under the Creati...
Immune cells and glia interact with neurons to alter pain sensitivity and to mediate the transition ...
Pain normally subserves a vital role in the survival of the organism, prompting the avoidance of sit...
AbstractInflammatory mediators act on peripheral sensory neurons to produce pain and hypersensitivit...
AbstractProstaglandin E2 (PGE2) and epinephrine act directly on nociceptors to produce mechanical hy...
Alterations in synaptic transmission within the spinal cord dorsal horn play a key role in the devel...
Clinical pain is previous termanext term serious public health issue. Treatment of pain-related suff...
Abstract: Tissue injury and inflammation result in release of various mediators that promote ongoing...
Clinical pain is a serious public health issue. Treatment of pain-related suffering requires knowled...
Chronic pain is a debilitating condition that influences the social, economic, and psychological asp...
Neck pain commonly results from whiplash injury and has a high incidence, with staggering annual cos...
Tissue injury, whether by trauma, surgical intervention, metabolic dysfunction, ischemia, or infecti...
Management of chronic pain is a real challenge, and current treatments focusing on blocking neurotra...
Pain is a complex phenomenon and the disentangling of the underlying mechanisms, in which peripheral...
Acute pain is protective and a cardinal feature of inflammation. Chronic pain after arthritis, nerve...
Copyright © 2015 Roberta Lattanzi et al. This is an open access article distributed under the Creati...
Immune cells and glia interact with neurons to alter pain sensitivity and to mediate the transition ...
Pain normally subserves a vital role in the survival of the organism, prompting the avoidance of sit...
AbstractInflammatory mediators act on peripheral sensory neurons to produce pain and hypersensitivit...
AbstractProstaglandin E2 (PGE2) and epinephrine act directly on nociceptors to produce mechanical hy...
Alterations in synaptic transmission within the spinal cord dorsal horn play a key role in the devel...
Clinical pain is previous termanext term serious public health issue. Treatment of pain-related suff...
Abstract: Tissue injury and inflammation result in release of various mediators that promote ongoing...
Clinical pain is a serious public health issue. Treatment of pain-related suffering requires knowled...
Chronic pain is a debilitating condition that influences the social, economic, and psychological asp...
Neck pain commonly results from whiplash injury and has a high incidence, with staggering annual cos...
Tissue injury, whether by trauma, surgical intervention, metabolic dysfunction, ischemia, or infecti...
Management of chronic pain is a real challenge, and current treatments focusing on blocking neurotra...
Pain is a complex phenomenon and the disentangling of the underlying mechanisms, in which peripheral...
Acute pain is protective and a cardinal feature of inflammation. Chronic pain after arthritis, nerve...
Copyright © 2015 Roberta Lattanzi et al. This is an open access article distributed under the Creati...
Immune cells and glia interact with neurons to alter pain sensitivity and to mediate the transition ...
Pain normally subserves a vital role in the survival of the organism, prompting the avoidance of sit...
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