Abstracts from the 44th Annual Meeting of the Society for Neuroscience in Anesthesiology and Critical Care, Chicago, IL, October 20–21, 2016
- Hermanides, Jeroen
- Hong, Young
- Trivedi, Monica
- Outtrim, Joanne
- Aigbirhio, Franklin
- Nestor, Peter
- Fryer, Timothy
- Menon, David
- Coles, Jonathan
DOIAbstract
Background: Metabolic derangements are common after traumatic brain injury (TBI) (Abate et al., Neurocrit Care, 2008), but few studies have combined 18F-fluorodeoxyglucose (FDG) and multi-tracer oxygen-15 (15O) positron emission tomography (PET) to interrogate underlying pathophysiological mechanisms (Hatori et al., J Nucl Med, 2004). We examined how derangements in FDG kinetic parameters within injured brain relate to cerebral blood flow (CBF), blood volume (CBV), oxygen metabolism (CMRO2) and oxygen extraction fraction (OEF)
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