<p>Model predictions of the effects of changes in the populations of susceptible and infected hosts on within-host pathogen diversity, over the host–pathogen population cycle. (A) The population size of uninfected hosts. (B) The population size of infectious cadavers (blue) and the mean nucleotide diversity (red).</p
<div><p>Pathogen transmission responds differently to host richness and abundance, two unique compon...
<p>We calculated daily human infectivity to mosquitoes, as a function of time post emergence, for 1,...
Pathogen transmission responds differently to host richness and abundance, two unique components of ...
<p>A) Pointwise prediction bands for the symptomatic infections produced by the model. B) The change...
The population dynamics of infectious disease is a mature field in terms of theory and to some exten...
The population dynamics of infectious disease is a mature field in terms of theory and to some exten...
This chapter explains why the traditional approach of predicting evolutionary outcomes by maximizing...
: Realistic population models have interactions between individuals. Such interactions cause populat...
Biological processes occur at distinct but interlinked scalesof organization. Yet, mathematical mode...
<p>Comparison of the predictions of our models (gray-shaded areas, showing 95% CIs of model realizat...
In this chapter we will model the mechanisms at the level of the individual infection process, inclu...
<p>The time evolution of the various singly (solid lines) and doubly (dashed lines) infected cell (l...
If viruses or other pathogens infect a single host, the outcome of infection may depend on the initi...
1. The hypothesis that multiple infections might disrupt or alter the density-dependent processes re...
<p>In all panels A–F, colored curves represent the pathogen population sizes of different virus stra...
<div><p>Pathogen transmission responds differently to host richness and abundance, two unique compon...
<p>We calculated daily human infectivity to mosquitoes, as a function of time post emergence, for 1,...
Pathogen transmission responds differently to host richness and abundance, two unique components of ...
<p>A) Pointwise prediction bands for the symptomatic infections produced by the model. B) The change...
The population dynamics of infectious disease is a mature field in terms of theory and to some exten...
The population dynamics of infectious disease is a mature field in terms of theory and to some exten...
This chapter explains why the traditional approach of predicting evolutionary outcomes by maximizing...
: Realistic population models have interactions between individuals. Such interactions cause populat...
Biological processes occur at distinct but interlinked scalesof organization. Yet, mathematical mode...
<p>Comparison of the predictions of our models (gray-shaded areas, showing 95% CIs of model realizat...
In this chapter we will model the mechanisms at the level of the individual infection process, inclu...
<p>The time evolution of the various singly (solid lines) and doubly (dashed lines) infected cell (l...
If viruses or other pathogens infect a single host, the outcome of infection may depend on the initi...
1. The hypothesis that multiple infections might disrupt or alter the density-dependent processes re...
<p>In all panels A–F, colored curves represent the pathogen population sizes of different virus stra...
<div><p>Pathogen transmission responds differently to host richness and abundance, two unique compon...
<p>We calculated daily human infectivity to mosquitoes, as a function of time post emergence, for 1,...
Pathogen transmission responds differently to host richness and abundance, two unique components of ...
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