Autoimmune-induced preferential depletion of myelin-associated glycoprotein (MAG) is genetically regulated in relapsing EAE (B6 × SJL) F1 mice-3

Levels of MAG (Fig. 1). Compared to control values, modest reduction of MBP levels was measured only in H-2mice during relapsing and chronic-sustained EAE. This antibody revealed presence of four distinct closely related 21–26 KD isoforms of MBP in control and EAE spinal cord, similarly as reported by the manufacturer of this antibody. These isoforms may be the result of posttranslational modification of MBP. Stripped and re-probed membrane was shown. Corresponding levels of GAPDH are shown in Fig. 6.<p><b>Copyright information:</b></p><p>Taken from "Autoimmune-induced preferential depletion of myelin-associated glycoprotein (MAG) is genetically regulated in relapsing EAE (B6 × SJL) F1 mice"</p><p>http://www.molecularneurodegeneration.com/content/3/1/7</p><p>Molecular Neurodegeneration 2008;3():7-7.</p><p>Published online 9 Jun 2008</p><p>PMCID:PMC2459167.</p><p>