BSO enhanced and sustained the DATS-induced cell cycle arrest but had little influence on DPTS-treated cells

HT-29 cells were pretreated with 500 μM BSO for 24 h and then the cells were treated with 10 μM DATS ( and ) or DPTS ( and ) for the times as indicated. The cell cycle distribution of the cells was analyzed by using a flow cytometer.<p><b>Copyright information:</b></p><p>Taken from "Alkenyl group is responsible for the disruption of microtubule network formation in human colon cancer cell line HT-29 cells"</p><p></p><p>Carcinogenesis 2008;29(7):1400-1406.</p><p>Published online 29 May 2008</p><p>PMCID:PMC2500214.</p><p>