Dexamethasone inhibits the HSV-/ ganciclovir bystander effect in malignant glioma cells-4

<p><b>Copyright information:</b></p><p>Taken from "Dexamethasone inhibits the HSV-/ ganciclovir bystander effect in malignant glioma cells"</p><p>BMC Cancer 2005;5():32-32.</p><p>Published online 2 Apr 2005</p><p>PMCID:PMC1080125.</p><p>Copyright © 2005 Robe et al; licensee BioMed Central Ltd.</p> + GCV and DEX + sFAS-L + GCV, assessed with a MTT test. This viability was significantly altered in cells treated with sFAS-L and GCV and this sensitization was abolished by DEX. Viability of LN18-TK cells after 24 hours of growth in the presence of GCV, sFAS-L, sFAS-L + GCV and DEX + sFAS-L + GCV. This viability was significantly altered in cells treated with sFAS-L and GCV and this effect was abolished by DEX. For both cell types, the viability in the absence of any drug was set as 100 percents. (*: < 0.05, ANOVA). Western blot analysis of the expression of CIAP-2, FAS/CD95 and BCLin C6, LN18 and U87 cells following treatment with DEX (1 μM)