<p>Western blot analysis of active cleaved caspase 3 (<b>A</b>), caspase 12 (<b>B</b>) and VEGF (<b>C</b>) in whole lung tissue homogenates. WB bands were normalized to beta-actin. **P<0.01; *P<0.05 when compared to a control group. Rats received a single i.p. injection of acrolein at 12 µmol/kg and were sacrificed 1, 2, 4 or 24 hours after the challenge. n = 4 animals per group, data are mean±S.E.</p
Background. Acrolein is a major component of environmental pollutants, cigarette smoke, and is also ...
Abstract—After 11 to 21 days, rats continuously exposed to 0.55 ppm acrolein had significantly lower...
Exposure of the airways to cigarette smoke (CS) is the primary risk factor for developing several lu...
<p>The VEGF (dimer and monomer) protein expression was normalized to beta-actin. *P<0.05; **P<0.01 w...
Given the relative abundance and toxic potential of acrolein in inhaled cigarette smoke, it is surpr...
Given the relative abundance and toxic potential of acrolein in inhaled cigarette smoke, it is surpr...
Fischer 344 rats were exposed to 0.0, 0.4, 1.4, or 4.0 ppm acrolein for 62 days. The major objective...
Background Given the relative abundance and toxic potential of acrolein in inhaled cigarette smoke, ...
Background Given the relative abundance and toxic potential of acrolein in inhaled cigarette smoke, ...
Nowadays, mitochondria are recognized as key players in the pathogenesis of a variety of smoking-ass...
Nowadays, mitochondria are recognized as key players in the pathogenesis of a variety of smoking-ass...
Nowadays, mitochondria are recognized as key players in the pathogenesis of a variety of smoking-ass...
Exposure to tobacco smoke, which contains several harmful and potentially harmful constituents such ...
<p><b>A.</b> Section of a representative control rat lung showing normal alveolar structure. <b>B.</...
<p>Overnight exposure to acrolein (ACR) induces IL-8 release from small airway epithelial cells (SAE...
Background. Acrolein is a major component of environmental pollutants, cigarette smoke, and is also ...
Abstract—After 11 to 21 days, rats continuously exposed to 0.55 ppm acrolein had significantly lower...
Exposure of the airways to cigarette smoke (CS) is the primary risk factor for developing several lu...
<p>The VEGF (dimer and monomer) protein expression was normalized to beta-actin. *P<0.05; **P<0.01 w...
Given the relative abundance and toxic potential of acrolein in inhaled cigarette smoke, it is surpr...
Given the relative abundance and toxic potential of acrolein in inhaled cigarette smoke, it is surpr...
Fischer 344 rats were exposed to 0.0, 0.4, 1.4, or 4.0 ppm acrolein for 62 days. The major objective...
Background Given the relative abundance and toxic potential of acrolein in inhaled cigarette smoke, ...
Background Given the relative abundance and toxic potential of acrolein in inhaled cigarette smoke, ...
Nowadays, mitochondria are recognized as key players in the pathogenesis of a variety of smoking-ass...
Nowadays, mitochondria are recognized as key players in the pathogenesis of a variety of smoking-ass...
Nowadays, mitochondria are recognized as key players in the pathogenesis of a variety of smoking-ass...
Exposure to tobacco smoke, which contains several harmful and potentially harmful constituents such ...
<p><b>A.</b> Section of a representative control rat lung showing normal alveolar structure. <b>B.</...
<p>Overnight exposure to acrolein (ACR) induces IL-8 release from small airway epithelial cells (SAE...
Background. Acrolein is a major component of environmental pollutants, cigarette smoke, and is also ...
Abstract—After 11 to 21 days, rats continuously exposed to 0.55 ppm acrolein had significantly lower...
Exposure of the airways to cigarette smoke (CS) is the primary risk factor for developing several lu...