Neither LuxS generated AI-2 signaling molecules or the AI-3 signaling system appeared to affect the growth and/or survival of hvKP1 in conditioned ascites.

<p>The growth of hvKP1 was assessed at 0, 3, 6, and 24 hours in human ascites (batch 2). <u>Panel A</u>: Ascites was not supplemented (unconditioned) or supplemented with hvKP1 generated conditioned ascites (final concentration 5%) or hvKP1Δ<i>luxS</i> generated conditioned ascites (batch 2) (final concentration 5%). There was no significant difference in the growth of hvKP1 when supplemented with either of these conditioned media. <u>Panel B</u>: Ascites was not supplemented (unconditioned) or supplemented with hvKP1 generated conditioned ascites (batch 2) (final concentration 5%) or epinephrine (final concentrations 50 µM and 500 µM). There was no significant difference in the growth of hvKP1 with the addition of either of these supplements. Data are Mean ± S.E.M.; N = 2–3.