<p>A to D, Representative tissue slide from remote myocardium with HE in sham (A), one week (B), four weeks (C) and eight weeks group (D); E, Accumulated results show myocytes size significantly increases four wks and eight wks post MI; F, Accumulated results show ANF, which is the hypertrophic factor, is up-regulated after MI. G, Accumulated results show myofilament density is significantly decreased eight weeks post MI (n = 4). # P<0.05 compared by group of 8 wks post MI. * P<0.05 compared by sham group.</p
Left ventricular (LV) remodeling following myocardial infarction (MI) is considered to contribute to...
Myocardial remodeling in response to chronic myocardial infarction (CMI) progresses through two phas...
thyrotoxic myocardium, myocyte composition. Several qualitative features of the ultrastructure of pr...
<p>A to D, Representative tissue slide from remote myocardium with vWF staining in sham (A), one wee...
Wall thickening (WT) is impaired in non-infarcted regions adjacent to Myocardial Infarction (MI) and...
<p>A. Photomicrographs of representative transverse sections of NLC CHF and Tg-CTGF CHF mice 4 weeks...
OBJECTIVES: To characterize the temporal alterations in native T1 and extracellular volume (ECV) of ...
To determine the compensatory reserve capacity of the ventricular myocardium following infarction, t...
Scarring and remodelling of the left ventricle (LV) after myocardial infarction (MI) results in isch...
Scarring and remodeling of the left ventricle (LV) after myocardial infarction (MI) results in ische...
For determination of the effects of myocardial infarction on the recovery potential of muscle mass i...
AbstractBackgroundThe time course and extent of recovery after revascularization of viable dysfuncti...
<p>The panels show the gene expression of ANP (<b>A</b>), BNP (<b>B</b>), β-myosin heavy chain (MHC)...
The contractile dysfunction of the hibernating myocardium in situ results from local environmental f...
<p>TNFR1 and TNFR2 gene deletion had opposite effects upon infarct size (A) and interstitial fibrosi...
Left ventricular (LV) remodeling following myocardial infarction (MI) is considered to contribute to...
Myocardial remodeling in response to chronic myocardial infarction (CMI) progresses through two phas...
thyrotoxic myocardium, myocyte composition. Several qualitative features of the ultrastructure of pr...
<p>A to D, Representative tissue slide from remote myocardium with vWF staining in sham (A), one wee...
Wall thickening (WT) is impaired in non-infarcted regions adjacent to Myocardial Infarction (MI) and...
<p>A. Photomicrographs of representative transverse sections of NLC CHF and Tg-CTGF CHF mice 4 weeks...
OBJECTIVES: To characterize the temporal alterations in native T1 and extracellular volume (ECV) of ...
To determine the compensatory reserve capacity of the ventricular myocardium following infarction, t...
Scarring and remodelling of the left ventricle (LV) after myocardial infarction (MI) results in isch...
Scarring and remodeling of the left ventricle (LV) after myocardial infarction (MI) results in ische...
For determination of the effects of myocardial infarction on the recovery potential of muscle mass i...
AbstractBackgroundThe time course and extent of recovery after revascularization of viable dysfuncti...
<p>The panels show the gene expression of ANP (<b>A</b>), BNP (<b>B</b>), β-myosin heavy chain (MHC)...
The contractile dysfunction of the hibernating myocardium in situ results from local environmental f...
<p>TNFR1 and TNFR2 gene deletion had opposite effects upon infarct size (A) and interstitial fibrosi...
Left ventricular (LV) remodeling following myocardial infarction (MI) is considered to contribute to...
Myocardial remodeling in response to chronic myocardial infarction (CMI) progresses through two phas...
thyrotoxic myocardium, myocyte composition. Several qualitative features of the ultrastructure of pr...