nNOSμ-deficiency does not affect the susceptibility of the TA to contraction-induced injury.

<p>Contraction-induced injury was produced by subjecting TA muscles to a series of consecutive lengthening contractions of progressively increasing strain. Strain is the percentage increase in length beyond optimal muscle length Lo. Contractile function is expressed as normalized force (force/initial force or P/Pi). The susceptibility to contraction-induced injury for wild type (black squares) and nNOS-deficient (grey diamonds) TA muscles are shown. There was no significant difference between controls and KN1 littermates. Numbers of animals analyzed: 14 wild type (8 males and 6 females) and 11 KN1 (5 males and 6 females) mice. Values shown are mean±standard error of the mean (S.E.M).