Acute, high concentration Cr(VI) treatment preferentially opens chromatin in promoters containing CTCF/BORIS and AP1 binding sites.

Misrepair of Particulate Chromium(VI)-Induced DNA Double Strand Breaks Leads to Neoplastic Transformation

Xie, Hong

May 2007

Hexavalent chromium (Cr(VI)) is considered a particularly important and dangerous environmental chem...

Characterizing the Role of Chromosome Instability in Particulate Hexavalent Chromium-induced Carcinogenesis

Wise, Sandra S

May 2013

Hexavalent chromium (Cr(VI)) compounds are established human lung carcinogens. Solubility plays a ke...

Binding of chromium(VI) to histones: Implications for chromium(VI)-induced genotoxicity

Levina, A.
Harris, H.
Lay, P.

January 2006

The original publication can be found at www.springerlink.comThe first evidence has been obtained fo...

Long-term low concentration Cr(VI) treatment preferentially opens chromatin in promoters containing AP1 binding sites.

Jerald L. Ovesen (565492)
Yunxia Fan (486948)
Xiang Zhang (19800)
Jing Chen (4762)
Mario Medvedovic (1779)
Ying Xia (222301)
Alvaro Puga (486949)

May 2014

HOMER analyses identified AP1 transcription factor binding motifs located within promoter element...

Long-term low dose and acute high dose Cr(VI) treatment affect chromatin structure very differently.

Jerald L. Ovesen (565492)
Yunxia Fan (486948)
Xiang Zhang (19800)
Jing Chen (4762)
Mario Medvedovic (1779)
Ying Xia (222301)
Alvaro Puga (486949)

May 2014

Top row: Venn diagrams of FAIRE DNA peaks induced in Hepa-1 cells treated with 25 µM Cr(VI...

Opening of FAIRE peaks in promoter regions by chromium treatment is significantly correlated with changes in gene expression.

Jerald L. Ovesen (565492)
Yunxia Fan (486948)
Xiang Zhang (19800)
Jing Chen (4762)
Mario Medvedovic (1779)
Ying Xia (222301)
Alvaro Puga (486949)

May 2014

(A) Venn diagram of concordant (p = 3.75×10−99) gene expression changes ...

Chromium disrupts chromatin organization and CTCF access to its cognate sites in promoters of differentially expressed genes

Andrew VonHandorf (4989422)
Francisco Javier Sánchez-Martín (486947)
Jacek Biesiada (438811)
Hongxia Zhang (164388)
Xiang Zhang (19800)
Mario Medvedovic (1779)
Alvaro Puga (486949)

May 2018

Hexavalent chromium compounds are well-established respiratory carcinogens used in industrial pro...

Formaldehyde-Assisted Isolation of Regulatory Elements (FAIRE) analysis uncovers broad changes in chromatin structure resulting from hexavalent chromium exposure.

Jerald L Ovesen
Yunxia Fan
Xiang Zhang
Jing Chen
Mario Medvedovic
Ying Xia
Alvaro Puga

The ability of chromatin to switch back and forth from open euchromatin to closed heterochromatin is...

Generation of S phase-dependent DNA double-strand breaks by Cr(VI) exposure

Linan Ha
Susan Ceryak
Steven R. Patierno

January 2004

Certain hexavalent chromium [Cr(VI)] compounds are implicated as occupational respiratory carcinogen...

Exposure to Chromium (VI) Enhances the Motility of Colorectal Cancer Cells through Activation of FAK

Chen, David

November 2017

Introduction Colorectal cancer (CRC) is the second leading cause of cancer-related death in the Un...

Impact of Carcinogenic Chromium on the Cellular Response to Proteotoxic Stress

Ferreira, Leonardo M. R.
Cunha-Oliveira, Teresa
Sobral, Margarida C.
Abreu, Patrícia L.
Alpoim, Maria Carmen
Urbano, Ana M.

October 2019

Worldwide, several million workers are employed in the various chromium (Cr) industries. These worke...

Carcinogenicity of hexavalent chromium

Holmes, Amie L.
Wise, S. S.
Wise, John P., Sr.

October 2008

Hexavalent chromium (Cr(VI)), a commonly used industrial metal, is a well known human lung carcinoge...

Complexities of chromium carcinogenesis: Role of cellular response, repair and recovery mechanisms

O\u27Brien, Travis J.
Ceryak, Susan
Patierno, Steven R.

December 2003

Certain hexavalent chromium (Cr(VI))-containing compounds are recognized occupational human lung car...

Comparison of Gene Expression Profiles in Chromate Transformed BEAS-2B Cells

Sun Hong
Clancy Harriet A.
Kluz Thomas
Zavadil Jiri
Costa Max

March 2011

Hexavalent chromium [Cr(VI)] is a potent human carcinogen. Occupational expo...

Elucidating a Mechanism of Particulate Hexavalent Chromium-Induced Carcinogenesis

Holmes, Amie L.

May 2011

Hexavalent chromium (Cr(VI)) compounds are well established human lung carcinogens. Solubility plays...

Misrepair of Particulate Chromium(VI)-Induced DNA Double Strand Breaks Leads to Neoplastic Transformation

Xie, Hong

May 2007

Hexavalent chromium (Cr(VI)) is considered a particularly important and dangerous environmental chem...

Characterizing the Role of Chromosome Instability in Particulate Hexavalent Chromium-induced Carcinogenesis

Wise, Sandra S

May 2013

Hexavalent chromium (Cr(VI)) compounds are established human lung carcinogens. Solubility plays a ke...

Binding of chromium(VI) to histones: Implications for chromium(VI)-induced genotoxicity

Levina, A.
Harris, H.
Lay, P.

January 2006

The original publication can be found at www.springerlink.comThe first evidence has been obtained fo...

Long-term low concentration Cr(VI) treatment preferentially opens chromatin in promoters containing AP1 binding sites.

Jerald L. Ovesen (565492)
Yunxia Fan (486948)
Xiang Zhang (19800)
Jing Chen (4762)
Mario Medvedovic (1779)
Ying Xia (222301)
Alvaro Puga (486949)

May 2014

HOMER analyses identified AP1 transcription factor binding motifs located within promoter element...

Long-term low dose and acute high dose Cr(VI) treatment affect chromatin structure very differently.

Jerald L. Ovesen (565492)
Yunxia Fan (486948)
Xiang Zhang (19800)
Jing Chen (4762)
Mario Medvedovic (1779)
Ying Xia (222301)
Alvaro Puga (486949)

May 2014

Top row: Venn diagrams of FAIRE DNA peaks induced in Hepa-1 cells treated with 25 µM Cr(VI...

Opening of FAIRE peaks in promoter regions by chromium treatment is significantly correlated with changes in gene expression.

Jerald L. Ovesen (565492)
Yunxia Fan (486948)
Xiang Zhang (19800)
Jing Chen (4762)
Mario Medvedovic (1779)
Ying Xia (222301)
Alvaro Puga (486949)

May 2014

(A) Venn diagram of concordant (p = 3.75×10−99) gene expression changes ...

Chromium disrupts chromatin organization and CTCF access to its cognate sites in promoters of differentially expressed genes

Andrew VonHandorf (4989422)
Francisco Javier Sánchez-Martín (486947)
Jacek Biesiada (438811)
Hongxia Zhang (164388)
Xiang Zhang (19800)
Mario Medvedovic (1779)
Alvaro Puga (486949)

May 2018

Hexavalent chromium compounds are well-established respiratory carcinogens used in industrial pro...

Formaldehyde-Assisted Isolation of Regulatory Elements (FAIRE) analysis uncovers broad changes in chromatin structure resulting from hexavalent chromium exposure.

Jerald L Ovesen
Yunxia Fan
Xiang Zhang
Jing Chen
Mario Medvedovic
Ying Xia
Alvaro Puga

The ability of chromatin to switch back and forth from open euchromatin to closed heterochromatin is...

Generation of S phase-dependent DNA double-strand breaks by Cr(VI) exposure

Linan Ha
Susan Ceryak
Steven R. Patierno

January 2004

Certain hexavalent chromium [Cr(VI)] compounds are implicated as occupational respiratory carcinogen...

Exposure to Chromium (VI) Enhances the Motility of Colorectal Cancer Cells through Activation of FAK

Chen, David

November 2017

Introduction Colorectal cancer (CRC) is the second leading cause of cancer-related death in the Un...

Impact of Carcinogenic Chromium on the Cellular Response to Proteotoxic Stress

Ferreira, Leonardo M. R.
Cunha-Oliveira, Teresa
Sobral, Margarida C.
Abreu, Patrícia L.
Alpoim, Maria Carmen
Urbano, Ana M.

October 2019

Worldwide, several million workers are employed in the various chromium (Cr) industries. These worke...

Carcinogenicity of hexavalent chromium

Holmes, Amie L.
Wise, S. S.
Wise, John P., Sr.

October 2008

Hexavalent chromium (Cr(VI)), a commonly used industrial metal, is a well known human lung carcinoge...

Complexities of chromium carcinogenesis: Role of cellular response, repair and recovery mechanisms

O\u27Brien, Travis J.
Ceryak, Susan
Patierno, Steven R.

December 2003

Certain hexavalent chromium (Cr(VI))-containing compounds are recognized occupational human lung car...

Comparison of Gene Expression Profiles in Chromate Transformed BEAS-2B Cells

Sun Hong
Clancy Harriet A.
Kluz Thomas
Zavadil Jiri
Costa Max

March 2011

Hexavalent chromium [Cr(VI)] is a potent human carcinogen. Occupational expo...

Elucidating a Mechanism of Particulate Hexavalent Chromium-Induced Carcinogenesis

Holmes, Amie L.

May 2011

Hexavalent chromium (Cr(VI)) compounds are well established human lung carcinogens. Solubility plays...

Misrepair of Particulate Chromium(VI)-Induced DNA Double Strand Breaks Leads to Neoplastic Transformation

Xie, Hong

May 2007

Hexavalent chromium (Cr(VI)) is considered a particularly important and dangerous environmental chem...

Characterizing the Role of Chromosome Instability in Particulate Hexavalent Chromium-induced Carcinogenesis

Wise, Sandra S

May 2013

Hexavalent chromium (Cr(VI)) compounds are established human lung carcinogens. Solubility plays a ke...

Binding of chromium(VI) to histones: Implications for chromium(VI)-induced genotoxicity

Levina, A.
Harris, H.
Lay, P.

January 2006

The original publication can be found at www.springerlink.comThe first evidence has been obtained fo...

Acute, high concentration Cr(VI) treatment preferentially opens chromatin in promoters containing CTCF/BORIS and AP1 binding sites.

Abstract

Extracted data

Acute, high concentration Cr(VI) treatment preferentially opens chromatin in promoters containing CTCF/BORIS and AP1 binding sites.

Abstract

Extracted data

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