<p>MPC (A) and MTT (B) were plated in vehicle (Ctr), 5 µM fluvastatin (Fluva), 5 µM simvastatin (Simva), 5 µM lovastatin (Lova) with or without 100 µM trans, trans farnesol (FOH) and spontaneous migration was recorded for 24.</p
AbstractObjective: Migration and proliferation of vascular smooth muscle cells (SMCs) contributes to...
The time dependence of lovastatin (LST) induced effects on proliferation and apoptosis were determin...
<p>(A) Cells were left untreated, or transfected to transiently express the HA-epitope-tagged ARF1 c...
<p>(A) HCT116/vector and HCT116/MACC1 cells were treated with 5 μM lovastatin for 24 h, and migratio...
<p>(A) Treatment with 0, 8 and 16 µm of simvastatin showed inhibited migration and invasion of A498 ...
Abstract Simvastatins are widely used to reduce endogenous cholesterol synthesis and improve hyperch...
<p>UMR-106 monolayer were scraped with a pipette tip and incubated in the absence or presence of sim...
Vascular smooth muscle cell (SMC) migration and proliferation contribute to the pathobiology of athe...
The beneficial effects of 3-hydroxy-3-methylglutaryl coenzyme A (HMG-CoA) reductase inhibitors (stat...
<p><b>A.</b> SW1736 cells were treated with troglitazone (5 μM) and/or lovastatin (1 μM) for 7 h, an...
<p>MPC (A–F) and MTT (G–K) were treated with ▪ 6.25 µM, ▴ 12.50 µM, ▾ 25.00 µM, and ⧫ 50.00 µM of at...
<p>(A) VSMC cells were cultured until near confluent in 6 well dishes. Cell migration was assessed u...
<p>xCelligence was used to monitor real-time cell migration. VSMCs starved of serum for 24 hours wer...
Statins are widely used hypocholesterolemic drugs that block the mevalonate pathway, responsible for...
<p>(A) After treating EPCs with 2.5–10 μM atorvastatin or rosuvastatin for 24 hours, an <i>in vitro<...
AbstractObjective: Migration and proliferation of vascular smooth muscle cells (SMCs) contributes to...
The time dependence of lovastatin (LST) induced effects on proliferation and apoptosis were determin...
<p>(A) Cells were left untreated, or transfected to transiently express the HA-epitope-tagged ARF1 c...
<p>(A) HCT116/vector and HCT116/MACC1 cells were treated with 5 μM lovastatin for 24 h, and migratio...
<p>(A) Treatment with 0, 8 and 16 µm of simvastatin showed inhibited migration and invasion of A498 ...
Abstract Simvastatins are widely used to reduce endogenous cholesterol synthesis and improve hyperch...
<p>UMR-106 monolayer were scraped with a pipette tip and incubated in the absence or presence of sim...
Vascular smooth muscle cell (SMC) migration and proliferation contribute to the pathobiology of athe...
The beneficial effects of 3-hydroxy-3-methylglutaryl coenzyme A (HMG-CoA) reductase inhibitors (stat...
<p><b>A.</b> SW1736 cells were treated with troglitazone (5 μM) and/or lovastatin (1 μM) for 7 h, an...
<p>MPC (A–F) and MTT (G–K) were treated with ▪ 6.25 µM, ▴ 12.50 µM, ▾ 25.00 µM, and ⧫ 50.00 µM of at...
<p>(A) VSMC cells were cultured until near confluent in 6 well dishes. Cell migration was assessed u...
<p>xCelligence was used to monitor real-time cell migration. VSMCs starved of serum for 24 hours wer...
Statins are widely used hypocholesterolemic drugs that block the mevalonate pathway, responsible for...
<p>(A) After treating EPCs with 2.5–10 μM atorvastatin or rosuvastatin for 24 hours, an <i>in vitro<...
AbstractObjective: Migration and proliferation of vascular smooth muscle cells (SMCs) contributes to...
The time dependence of lovastatin (LST) induced effects on proliferation and apoptosis were determin...
<p>(A) Cells were left untreated, or transfected to transiently express the HA-epitope-tagged ARF1 c...