Stifffness-dependent glioma cell proliferation is dampened upon treatment with 20 uM EGFR inhibitor - Tyrphostin, 20 uM Akt inhibitor - Triciribine, and 20 uM PI3 Kinase inhibitor - Wortmannin for 24 hours as compared with the DMSO negative control.

<p>Results represent quantification of n>10,000 cells for at least three substrates per condition by flow cytometry, where the percentage of dividing cells was determined as the average percentage of cells staining positive for BrdU incorporation *, P<0.05 with respect to 119 kPa for DMSO control, 20 uM Tyrphostin and 20 uM Triciribine.