Several examples of genomic changes other than essential splice-site mutations causing splicing aberrations obtained from our comparative whole genome and transcriptome sequencing analyses.
- Yuichi Shiraishi (677117)
- Akihiro Fujimoto (273946)
- Mayuko Furuta (395068)
- Hiroko Tanaka (677118)
- Ken-ichi Chiba (677119)
- Keith A. Boroevich (540233)
- Tetsuo Abe (677120)
- Yoshiiku Kawakami (128669)
- Masaki Ueno (121130)
- Kunihito Gotoh (677121)
- Shun-ichi Ariizumi (677122)
- Tetsuo Shibuya (677123)
- Kaoru Nakano (677124)
- Aya Sasaki (228698)
- Kazuhiro Maejima (677125)
- Rina Kitada (9338)
- Shinya Hayami (677126)
- Yoshinobu Shigekawa (677127)
- Shigeru Marubashi (677128)
- Terumasa Yamada (677129)
- Michiaki Kubo (128298)
- Osamu Ishikawa (585936)
- Hiroshi Aikata (128663)
- Koji Arihiro (56456)
- Hideki Ohdan (300684)
- Masakazu Yamamoto (588188)
- Hiroki Yamaue (277099)
- Kazuaki Chayama (128675)
- Tatsuhiko Tsunoda (128303)
- Satoru Miyano (59616)
- Hidewaki Nakagawa (128309)
Publication date
December 2014
DOIAbstract
<p>Exonic and intronic sequences are designated by capital and small letters, respectively. Red sequences are somatic mutations in HCCs. Blue and green numbers on the side of sequences are edit distances from splicing donor motif (AG|GTRAGT, <a href="http://www.plosone.org/article/info:doi/10.1371/journal.pone.0114263#pone.0114263-Zhuang1" target="_blank">[38]</a>) and splicing acceptor motif (YYYYNCAG|G), respectively. Most somatic mutations changed the edit distance to splicing donor motifs so that the corresponding alteration can be enhanced.</p
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