<p>(A) As seen by periodic acid-Schiff (PAS) staining, the tubular injury was partially restored from day 3 to day 28 (D3 to D28). (B) As seen by Masson’s trichrome (MT) staining, we observed the development and progression of interstitial fibrosis in the kidneys during the 28 days. (n = 5–6 per group), (C) In immunohistochemical staining, we detected the upregulation of TGF-β. Magnification: ×100, (n = 4–6 per group), *p < 0.05 compared to day 0, <sup>†</sup>p < 0.05 compared to the previous time point.</p
Abstract Background To establish a model of chronic renal fibrosis following acute kidney injury (AK...
<p>Absence of glomerular or tubular injury in kidney sections of rats in SO-P (A) and SO-T3 (B) grou...
<p>(A) Representative histological findings in tissue stained with Masson’s trichrome 7 days after s...
<p>(A) Representative images of hematoxylin and eosin-stained kidney sections from five animals in e...
<p>Immunohistochemical staining of representative kidney sections from different experimental groups...
Ischemic acute renal failure: Long-term histology of cell and matrix changes in the rat.BackgroundTh...
<p><b>(A)</b> Representative light microscopy images of HE-stained kidney sections from the Sham and...
<p>(A) Varying degrees of renal pathological changes from 1 h to 24 h after reperfusion in the IRI g...
Figure S2. Histological evaluation of fibroblast marker in the ischemic kidneys. UIRI was performed ...
<p>A, B, C = Renal cortex at 3 hours (h) post ischemia-reperfusion (IR); D, E, F = Renal outer medul...
Persistent renal and extrarenal immune changes after severe ischemic injury.BackgroundRenal ischemia...
<p>PAS staining of representative kidney sections from different experimental groups. (A) The epithe...
Acute kidney injury (AKI) is an underestimated, yet important risk factor for development of chronic...
<p>Mice were subjected to 45 min renal ischemia to the right kidney and then allowed to recover for ...
<p>C57BL/6 mice were subjected to sham or bilateral ischemia by clamping the renal pedicles for 30 m...
Abstract Background To establish a model of chronic renal fibrosis following acute kidney injury (AK...
<p>Absence of glomerular or tubular injury in kidney sections of rats in SO-P (A) and SO-T3 (B) grou...
<p>(A) Representative histological findings in tissue stained with Masson’s trichrome 7 days after s...
<p>(A) Representative images of hematoxylin and eosin-stained kidney sections from five animals in e...
<p>Immunohistochemical staining of representative kidney sections from different experimental groups...
Ischemic acute renal failure: Long-term histology of cell and matrix changes in the rat.BackgroundTh...
<p><b>(A)</b> Representative light microscopy images of HE-stained kidney sections from the Sham and...
<p>(A) Varying degrees of renal pathological changes from 1 h to 24 h after reperfusion in the IRI g...
Figure S2. Histological evaluation of fibroblast marker in the ischemic kidneys. UIRI was performed ...
<p>A, B, C = Renal cortex at 3 hours (h) post ischemia-reperfusion (IR); D, E, F = Renal outer medul...
Persistent renal and extrarenal immune changes after severe ischemic injury.BackgroundRenal ischemia...
<p>PAS staining of representative kidney sections from different experimental groups. (A) The epithe...
Acute kidney injury (AKI) is an underestimated, yet important risk factor for development of chronic...
<p>Mice were subjected to 45 min renal ischemia to the right kidney and then allowed to recover for ...
<p>C57BL/6 mice were subjected to sham or bilateral ischemia by clamping the renal pedicles for 30 m...
Abstract Background To establish a model of chronic renal fibrosis following acute kidney injury (AK...
<p>Absence of glomerular or tubular injury in kidney sections of rats in SO-P (A) and SO-T3 (B) grou...
<p>(A) Representative histological findings in tissue stained with Masson’s trichrome 7 days after s...