Background: The effectiveness and limitations of causal therapy of chronic periodontitis have been thoroughly documented in the literature, and the intensity of the individual destructive host response is taken into consideration, as well. These facts are the basis in research of additional therapeutic approaches to modify tissue response by regulating the amount and activity of pro-inflammatory mediators and those of destruction. Various drugs are being studied to modulate the host's response in chronic periodontitis. The use of non-steroidal anti-inflammatory drugs that inhibit the expression and activity of important mediators has led to positive results. Aim: To assess changes in PGE2 gene expression levels as a result of non-surgical ...
AIM: To describe the kinetics of serum inflammatory markers after a course of treatment comprising s...
The goal of periodontal therapy includes management of bacterial pathogens to control the destructio...
Introduction: The receptor activator of NF-kB ligand (RANKL) and osteoprotegerin (OPG) are molecules...
Background: The understanding of the pathogenesis of periodontitis makes various progresses in the ...
Background: The understanding of the pathogenesis of periodontitis makes various progresses in the ...
PubMed ID: 12593597Background: The purpose of the present study was to evaluate the effect of a rela...
Background: Nonsteroidal anti‑inflammatory drugs (NSAIDs) are widely used for inflammation control a...
WOS: 000187314000008PubMed ID: 12593597Background: The purpose of the present study was to evaluate ...
Abstract: The systemic effect of chronic periodontitis (CP) has been suggested by several studies as...
We aimed to evaluate the impact of non-surgical periodontal treatment on the salivary expression of ...
Aim: To describe the kinetics of serum inflammatory markers after a course of treatment comprising s...
The purpose of this analysis of inflammation markers from a double blinded, randomized, controlled c...
AIM: To describe the kinetics of serum inflammatory markers after a course of treatment comprising s...
Objective: To assess whether non-surgical periodontal treatment is associated with changes in serolo...
AIM: To describe the kinetics of serum inflammatory markers after a course of treatment comprising s...
AIM: To describe the kinetics of serum inflammatory markers after a course of treatment comprising s...
The goal of periodontal therapy includes management of bacterial pathogens to control the destructio...
Introduction: The receptor activator of NF-kB ligand (RANKL) and osteoprotegerin (OPG) are molecules...
Background: The understanding of the pathogenesis of periodontitis makes various progresses in the ...
Background: The understanding of the pathogenesis of periodontitis makes various progresses in the ...
PubMed ID: 12593597Background: The purpose of the present study was to evaluate the effect of a rela...
Background: Nonsteroidal anti‑inflammatory drugs (NSAIDs) are widely used for inflammation control a...
WOS: 000187314000008PubMed ID: 12593597Background: The purpose of the present study was to evaluate ...
Abstract: The systemic effect of chronic periodontitis (CP) has been suggested by several studies as...
We aimed to evaluate the impact of non-surgical periodontal treatment on the salivary expression of ...
Aim: To describe the kinetics of serum inflammatory markers after a course of treatment comprising s...
The purpose of this analysis of inflammation markers from a double blinded, randomized, controlled c...
AIM: To describe the kinetics of serum inflammatory markers after a course of treatment comprising s...
Objective: To assess whether non-surgical periodontal treatment is associated with changes in serolo...
AIM: To describe the kinetics of serum inflammatory markers after a course of treatment comprising s...
AIM: To describe the kinetics of serum inflammatory markers after a course of treatment comprising s...
The goal of periodontal therapy includes management of bacterial pathogens to control the destructio...
Introduction: The receptor activator of NF-kB ligand (RANKL) and osteoprotegerin (OPG) are molecules...