Journal of Inflammation Research Knockout of the c-Jun N-terminal Kinase 2 aggravates the development of mild chronic dextran sulfate sodium colitis independently of expression of intestinal cytokines TNFα, TGFB1, and IL-
Previous work has suggested that the LIGHT-TR2 costimulatory pathway plays a role in the acute and c...
Inflammatory bowel diseases (IBD), including mainly ulcerative colitis (UC) and Crohn's disease (CD)...
Ulcerative colitis (UC) is characterized bychronic inflammation of the colonic mucosa. Administratio...
Journal of Inflammation Research Knockout of the c-Jun N-terminal Kinase 2 aggravates the developmen...
Sabine Kersting,1 Kirstin Reinecke,2 Christoph Hilgert,1 Monika S Janot,1 Elisabeth Haarmann,1 Marti...
AbstractInflammatory bowel disease (IBD) affects millions of people worldwide but its pathophysiolog...
The c-Jun N-terminal kinase (JNK)-inhibiting peptide D-JNKI-1, syn. XG-102 was tested for its therap...
Tumor necrosis factor-a (TNF-a) is a key factor for the pathogenesis of inflammatory bowel diseases ...
textabstractThe object of this study was to establish whether different pro- and anti-inflammatory m...
Die Entstehung der chronisch entzündlichen Darmerkrankungen ist ein multifaktorieller Prozess, in de...
Abstrect Background To understand whether TLR-4-linked NF-kB activation negatively correlates with l...
Calcitriol analog ZK191784 ameliorates acute and chronic dextran sodium sulfate-induced colitis by m...
BACKGROUND: The proinflammatory cytokines interleukin 1beta (IL-1beta) and IL-18 are central players...
Background: The role of integrin signaling in mucosal inflammation is presently unk...
Uncontrolled chronic inflammation and necrosis is characteristic of inflammatory bowel disease (IBD)...
Previous work has suggested that the LIGHT-TR2 costimulatory pathway plays a role in the acute and c...
Inflammatory bowel diseases (IBD), including mainly ulcerative colitis (UC) and Crohn's disease (CD)...
Ulcerative colitis (UC) is characterized bychronic inflammation of the colonic mucosa. Administratio...
Journal of Inflammation Research Knockout of the c-Jun N-terminal Kinase 2 aggravates the developmen...
Sabine Kersting,1 Kirstin Reinecke,2 Christoph Hilgert,1 Monika S Janot,1 Elisabeth Haarmann,1 Marti...
AbstractInflammatory bowel disease (IBD) affects millions of people worldwide but its pathophysiolog...
The c-Jun N-terminal kinase (JNK)-inhibiting peptide D-JNKI-1, syn. XG-102 was tested for its therap...
Tumor necrosis factor-a (TNF-a) is a key factor for the pathogenesis of inflammatory bowel diseases ...
textabstractThe object of this study was to establish whether different pro- and anti-inflammatory m...
Die Entstehung der chronisch entzündlichen Darmerkrankungen ist ein multifaktorieller Prozess, in de...
Abstrect Background To understand whether TLR-4-linked NF-kB activation negatively correlates with l...
Calcitriol analog ZK191784 ameliorates acute and chronic dextran sodium sulfate-induced colitis by m...
BACKGROUND: The proinflammatory cytokines interleukin 1beta (IL-1beta) and IL-18 are central players...
Background: The role of integrin signaling in mucosal inflammation is presently unk...
Uncontrolled chronic inflammation and necrosis is characteristic of inflammatory bowel disease (IBD)...
Previous work has suggested that the LIGHT-TR2 costimulatory pathway plays a role in the acute and c...
Inflammatory bowel diseases (IBD), including mainly ulcerative colitis (UC) and Crohn's disease (CD)...
Ulcerative colitis (UC) is characterized bychronic inflammation of the colonic mucosa. Administratio...