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Acquired maternal infection with Toxoplasma gondii (T. gondii) during pregnancy may cause congenital toxoplasmosis. Transplacentally acquired infection of the fetus may cause chorioretinitis, severe thrombocyto-paenia, intracranial calcification, hepatosplenomegaly, and disturbances of head size, etc. In infected newborns, who appear normal at birth, retinal scars may develop slowly during the first 3–4 years of life, either with or without accompanying symptoms. The retinal lesion is probably the most frequent manifestation of congenital toxoplasmosis.1–3 If the retinal damage occurs in the macular area, blindness may result. Each case of con-genital toxoplasmosis is a tragic event for the family and a social burden to society. Prevention of congenital toxoplasmosis is a step