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d form lative college students in the United States (Wechsler et al., 1995). Binge drinking has been linked to a range of antisocial behaviors and other forms of psychopathology, including 1 gene (CRHR1) and binge drinking, lifetime prevalence of alcohol use, and lifetime prevalence of alcohol intoxica-tion. Herman et al. (2003) found a significant association between the serotonin transporter promoter polymorphism 5-HTTLPR (SLC6A4) and frequency of binge drinking, * Corresponding author. Tel.: þ1-314-977-2718. (2009According to the National Institute on Alcohol Abuse and Alcoholism, binge drinking is a pattern of drinking that brings a person’s blood alcohol concentration to 0.08 g percent or above, which typically occurs when males consume five or more drinks and females consume four or more drinks over a two-hour span. Like most forms of antisocial behavior, binge drinking is more prevalent among males than females and among those in late adolescence and early adulthood. A national epidemiologic study found that males accounted for 81%of binge drinking episodes and young adults between the ages of 18 and 25 accounted for nearly one-third of all binge drinking (Naimi et al., 2003). The national lifetime prevalence of binge drinking in the United States is estimated to be17%(Townet al., 2006) and affectsmore than sixmillion 2005). Using data from Russian respondents, Pridemore (2004) found links between binge drinking and violent criminal offenses, including homicide. The co-occurrence of binge drinking with other substance abuse and antisocial behaviors is suggestive of a general propensity that likely has a partially genetic etiology. A number of genes have been linked to alcohol depen