Add to ORKGPENTAPEPTIDE REPEAT PROTEINS..................................................................................................................665 Discovery of qnr Genes...........................................................................................................................................665 qnrA.......................................................................................................................................................................665 qnrS.......................................................................................................................................................................66
Fluoroquinolone resistance in Enterobacteriales is developed by chromosomal and plasmid-mediated mec...
Background/Purpose: Only limited information is available about the detailed characteristics of qnrD...
Quinolones are widely used antimicrobial agents, particularly for the treatment of infections caused...
Objectives: To study the role of Qnr-like pentapeptide repeat proteins (PRPs) from several Gram-posi...
Although quinolone resistance results mostly from chromosomal mutations in Enterobacteriaceae, it ma...
Quinolones are synthetic and commonly used antibiotics for treatment of multiple clinical infections...
Background: Plasmid-encoded quinolone resistance protein Qnr is an important factor in bacterial res...
Objectives: A new pentapeptide repeat (PRP) protein, named SmaQnr, from the clinically relevant spec...
AbstractAlthough resistance to quinolones is commonly chromosomally-encoded in Enterobacteriaceae, t...
Background: Fluoro-/Quinolones are important antimicrobial substances for treatment of animal and hu...
ABSTRACTThis study investigated the effect of copy number and transcriptional level of the qnrA gene...
qnr genes were discovered on plasmids by their ability to reduce quinolone susceptibility, but homol...
喹诺酮类药物(Quinolones)是一类广谱、强效的化学合成抗细菌药物.长期以来,细菌通过染色体介导的靶位点改变[1]、蓄积减少[2](包括孔蛋白缺失、主动外排增加)等机制逐渐对其形成耐药.中文核心...
Bacteria develop resistance to antimicrobial agents by a number of different mechanisms. The resista...
Resistance to quinolones and fluoroquinolones is being increasingly reported among human but also ve...
Fluoroquinolone resistance in Enterobacteriales is developed by chromosomal and plasmid-mediated mec...
Background/Purpose: Only limited information is available about the detailed characteristics of qnrD...
Quinolones are widely used antimicrobial agents, particularly for the treatment of infections caused...
Objectives: To study the role of Qnr-like pentapeptide repeat proteins (PRPs) from several Gram-posi...
Although quinolone resistance results mostly from chromosomal mutations in Enterobacteriaceae, it ma...
Quinolones are synthetic and commonly used antibiotics for treatment of multiple clinical infections...
Background: Plasmid-encoded quinolone resistance protein Qnr is an important factor in bacterial res...
Objectives: A new pentapeptide repeat (PRP) protein, named SmaQnr, from the clinically relevant spec...
AbstractAlthough resistance to quinolones is commonly chromosomally-encoded in Enterobacteriaceae, t...
Background: Fluoro-/Quinolones are important antimicrobial substances for treatment of animal and hu...
ABSTRACTThis study investigated the effect of copy number and transcriptional level of the qnrA gene...
qnr genes were discovered on plasmids by their ability to reduce quinolone susceptibility, but homol...
喹诺酮类药物(Quinolones)是一类广谱、强效的化学合成抗细菌药物.长期以来,细菌通过染色体介导的靶位点改变[1]、蓄积减少[2](包括孔蛋白缺失、主动外排增加)等机制逐渐对其形成耐药.中文核心...
Bacteria develop resistance to antimicrobial agents by a number of different mechanisms. The resista...
Resistance to quinolones and fluoroquinolones is being increasingly reported among human but also ve...
Fluoroquinolone resistance in Enterobacteriales is developed by chromosomal and plasmid-mediated mec...
Background/Purpose: Only limited information is available about the detailed characteristics of qnrD...
Quinolones are widely used antimicrobial agents, particularly for the treatment of infections caused...